Mr. Sunday Igboho write FG, says He’s not at war with anyone.

The Nigeria Yoruba Nation activist, Mr. Sunday Adeyemo, popularly known as Sunday Igboho, has stated that he is not at war with the President Muhammadu Buhari, state governors or any government official.

Mr. Sunday Igboho disclosed this in his 2022 New Year Message signed by one of his lawyers in Nigeria, Pelumi Olajengbesi.

The activist said everyone living in the country deserves to be protected regardless of their tribe, religion or political leanings.

The message reads, “Let it be abundantly clear that I am not at war with President Buhari, State Governors or anybody in government. What I want the government to do is address the genesis of my agitation for a Yoruba Nation. The genesis is the insecurity in Ibarapaland and other places in the South-West where farmers are habitually maimed, hacked to death, kidnapped and raped as if there are no security agents in the country. Everybody deserves to be protected and ought to feel safe in their country regardless of their tribe, religion or political leaning.

 “At some point, our people became helpless and could no longer go to their farmlands again. Scores of farmers were butchered to death in 2021 and the government pretended that all was okay. No, this must not continue and this was why I rose to stop the menace. I did not speak up to fight government, I spoke up to ensure the protection of my people and land.

“In 2022, the government must rise up and put an end to the havoc caused by herdsmen that are criminals and give everyone a sense of belonging whether foreigners or locals and stop the attack on my people in Yorubaland.

I am not selfish and this has never been about me. I am more concerned about the helpless citizens who buried their loved ones to aimless killings in 2021 and I pray that the Almighty God through divine intervention stops the killings of innocent people in Nigeria in 2022 and beyond. 

In 2022, it would indeed be to my greatest satisfaction to see my people live without fear and with the confidence that their lives matter if no place else then in their own homes, lands and farms. This is my abiding hope for my people, so help me God.”

For over five months, the 49-year-old agitator has been in detention in neighbouring Benin Republic since his arrest around 8pm on July 19, 2021 at the Cadjèhoun Airport in Cotonou.

The Cour De’appal De Cotonou since the arraignment of Igboho on July 27, 2021, has not sat, fuelling speculations that the detention of Igboho is laced with political undertones.

The Department of State Services (DSS) had on July 1st raided the residence of the activist in Ibadan, Oyo State.

Chief Igboho had planned to attend a Yoruba Nation Rally in Lagos before the raid.

DSS raided the residence, killing two people and arresting 12 aides but Igboho escaped.

DSS alleged that Igboho was stockpiling weapons, and subsequently declared him wanted, but he has since denied the allegation. 


Antarctica, the southernmost continent and site of the South Pole, is a virtually uninhabited, ice-covered landmass.

Antarctica view point

Antarctica, the southernmost continent and site of the South Pole, is a virtually uninhabited, ice-covered landmass. Most cruises to the continent visit the Antarctic Peninsula, which stretches toward South America.

It’s known for the Lemaire Channel and Paradise Harbor, striking, iceberg-flanked passageways, and Port Lockroy, a former British research station turned museum. The peninsula’s isolated terrain also shelters rich wildlife, including many penguins.

Antarctica view.

Fast facts

  • Antarctica is the highest, driest, coldest and windiest continent on Earth
  • Antarctica covers 14.2 million km² (5.5 million square miles)
  • The Antarctic ice sheet is the largest ice store on earth
    • Area: 5.4 million square mile (14 million kilometres)
    • Mass: 7.2 million cubic miles (30 million cubic metres)
    • Maximum depth: 15,669 feet (4776 metres)
    • Average depth: 7000 feet (2160 metres)
    • Covers roughly 98% of Antarctica
    • Contains 90% of the ice on earth
    • Contains 70% of the world’s freshwater.

. Antarctica holds most of the world’s fresh water

An incredible 60-90% of the world’s fresh water is locked in Antarctica’s vast ice sheet. The Antarctic ice sheet is the largest on Earth, covering an incredible 14 million km² (5.4 million square miles) of Antarctic mountain ranges, valleys and plateaus. This leaves only 1% of Antarctica permanently ice-free. Some areas are ice-free in the summer, including many of the areas we visit on the Antarctic Peninsula.

At its deepest, Antarctica’s ice is 4.5km (2.7 miles) thick – that’s half the height of Mt Everest! If it all melted, global sea levels would rise about 60 m (200 ft). 

Antarctica ice view

2. Antarctica is a desert

With all of that fresh water held in the ice sheet, how could Antarctica be a desert?

When most of us think of deserts we think of sand dunes and sizzling temperatures, but technically a desert doesn’t have to be hot or sandy, it’s more about how much precipitation the area receives as rain, snow, mist or fog. A desert is any region that receives very little annual precipitation.

The average annual rainfall at the South Pole over the past 30 years was just over 10 mm (0.4 in). Although there is more precipitation towards the coast, the average across the continent is low enough to classify Antarctica as a polar desert.

Antarctica view

So while Antarctica may be covered in ice, it has taken an incredible 45 million years to grow to its current thickness, because so little rain falls there.

As well as being one of the driest continents on Earth, Antarctica is also the coldest, windiest and highest.


Kaduna State Government has shut down two Islamiyah schools over sexual abuse of minors.

Oneworldnews line revealed that the Kaduna State Schools Quality Assurance Authority (KSSQA) has shut down two Islamiyah schools over the alleged sexual abuse of minors.

According to a Vanguard report, one of the schools, Madrasatul Ulumul Deeniya wa Tahfizul Qur’an, located in Rigasa, was closed over the alleged rape of a six-year-old girl in the school premises.

Explaining the reason for the closure of the schools, the overseer of the KSSQA, Idris Aliyu, said the six-year-old girl was violated within the school premises and the school authority covered it up and claimed ignorance of the incident.

The other Islamiyah school, located at Tsohon Masallacin Juma’a in Kachia, Kachia Local Government Area of Kaduna State was shut after a 50-year-old teacher at the school allegedly impregnated a 12-year-old student.

Speaking further on the case of the 6-year-old, Aliyu said information before the Ministry of Education showed that the survivor’s grandmother went to the school authority and loaded a complaint but was allegedly beaten by the students and teachers of the Islamiyah.

He said that Governor Nasir El-Rufai has ordered that the Islamiyah be shut down indefinitely until an investigation was concluded and the culprit apprehended.

The headmaster of the school would be held responsible for the act until all persons in the school were profiled and the perpetrator identified by the survivor and prosecuted.

The individuals responsible for the violence against the grandmother, especially the headmaster, will also be prosecuted.

This is in line with the state government’s responsibility in protecting the vulnerable population in the state,” he said..

The headteacher of the school, Malam Kabir Abdullahi, denied the allegation, saying “it is not a rape. What we gathered is that an unidentified boy injured the girl in her private parts with a stick, he said.

Abdullahi said that the case was already being investigated by the Divisional Police Officer (DPO), Rigasa, Abubakar Bauranya.

Meanwhile, the DPO confirmed receiving the case and equally aligned with the narrative of the headteacher of the Islamiyah school.

But the grandmother of the survivor, Hajiya Batul Gambo insisted that her grandchild was violated and had been confirmed by Sexual Assault Referral Centre, Yusuf Dantsoho Memorial Hospital Tudun Wada, Kaduna.

A medical report signed by Dr A Raji confirmed Gambo’s claim that the six-year-old was raped. 

Raji’s report attributed the bleeding to virginal bruising of the labia minora, bilaterally per urethral bruising, heamatoma of the clitoris and laceration of the hymen.

Commenting on the rape and impregnation of the 12-year-old girl in Kachia, Aliyu said that the survivor was already six months pregnant.

He said that the school was equally closed as directed by El-Rufai, stressing that the safety of the pupils cannot be guaranteed.

He explained that the Commissioner of Human Services and Social Development, Hajiya Hafsat Baba and the Commissioner for Education, Hajiya Halima Lawal would work closely with the police to investigate and ensure justice is done.

Meanwhile, the two schools will remain closed until investigations are concluded and the culprit prosecuted in accordance with the law, he said.


Ten persons dead in a clash between the Nigeria Navy and kidnappers in Anambra State.

The Special Force Squad of the Nigerian Navy Base, Onitsha, has stormed the Ochan forest,the Anambra State base of kidnappers, arrested five suspected kidnappers, and killed over 10 while others escaped with bullet wounds.

Parading the suspects at its base in Onitsha on Friday, the leader of the naval team, Suleiman Agabi, said the command, based on intelligence gathering, stormed the kidnappers hideouts at about 1:20 am on Friday.

Agabi confirmed that the suspected kidnappers were the masterminds behind the abduction of the traditional ruler of Ogwaniocha community in Anambra State, Igwe Oliver Nnaji, who was kidnapped on November 15, 2021.

The suspects are making confessional statements in connection to the abduction of the monarch.


Arrested notorious bandit leader begs for forgiveness.

According to Oneworld news line the elements of banditry who have made both the North-Western and some parts of North-Central Nigeria war zones are dangerous, heartless and inhuman.

They have not even a single iota of pity for the human life. And with their level of wickedness, it has become a question that begs for an answer if their Boko Haram and ISWAP terrorist counterparts are really more dangerous than they are.

It was in the light of this that Nigerians of all walks of life, and both within the country and the diaspora, had to mount pressure on the federal government (FG) to declare them terrorists like the foregoing two groups in order that they may be thoroughly tackled as well.

This is why the judgement that was pasef by Justice Taiwo Taiwo of the Abuja-based federal high court in which he eventually pronounced them so on the 26th of November was a commendable one.

The recently proscribed bandits, like every other normal association of people, also have leadership echelons, albeit they are scattered all around the forests where they operate from. Thus, they have overall leaders, their assistants and others.

One of such so-called leaders was a certain Alhaji Goma Sama’ila who was reportedly captured on the night of Friday 24th of September in Kaduna State.

Before his arrest, the Alhaji bandit leader was said to be rated among the categories of the likes of Turji Kachalla, Halilu Sububu and the now late Dogo Gide as the most dreadful and wanted criminals in the country.

That was because of their notoriety in their banditry activities in Zamfara, Kaduna and Katsina States where they allegedly indulge in kidnapping, cattle rustling, armed robbery and destructions of lives and property.

However, in his confessions upon his apprehension, after admitting his culpability to the crimes, Sama’ila went on to plead for mercy, and that he would not go back to criminal ways if he should be forgiven.

And according to him, he had said the truth because whoever said the truth is one in accord with Allah (God).


Adrenomyeloneuropathy (AMN) is an inherited condition that affects the spinal cord. It is a form of X-linked adrenoleukodystrophy.


Adrenomyeloneuropathy (AMN) is an inherited condition that affects the spinal cord. It is a form of X-linked adrenoleukodystrophy. On average, people with AMN begin to develop features in the late twenties. Signs and symptoms may include progressive stiffness and weakness of the legs; ataxia; speech difficulties; adrenal insufficiency; sexual dysfunction; and bladder control issues. Some people with AMN also have brain involvement which can lead to behavioral abnormalities, vision loss, hearing problems, and/or seizures. AMN is caused by changes (mutations) in the ABCD1 gene and is inherited in an X-linked manner. Treatment addresses the symptoms in each person and may include steroid replacement therapy for adrenal insufficiency.


Adrenomyeloneuropathy (AMN) is inherited in an X-linked manner.[4] A condition is considered X-linked if the responsible gene is located on the X chromosome, one of the two sex chromosomes. The Y chromosome is the other sex chromosome. Women have two X chromosomes and men have an X and a Y chromosome.

Because men have one copy of the X chromosome, a mutated copy of the responsible gene in each cell is enough to cause signs or symptoms of the condition. A man with an X-linked condition will pass the mutation to all of his daughters and none of his sons.

Because women have two X chromosomes, the effect of the mutation may be masked by the other, normal copy of the gene, on the other X chromosome. Women with one mutation are often referred to as “carriers” of an X-linked condition. In most cases, carrier women have no symptoms or are mildly affected. However, some may be just as severely affected as males with the condition. Women who carry an X-linked condition have a 50% chance of passing the mutation on with each pregnancy.

In AMN approximately 20% of women who carry one ABCD1 mutation will develop progressive stiffness and weakness in their legs, often in middle age or later. These women typically have normal adrenal function.

What Causes AMN?

As with ALD, AMN is caused by genetically inherited mutations to the gene ABCD1. ABCD1 normally encodes the protein adrenoleukodystrophy protein (ALDP) which is involve in the transport of very long chain fatty acids (VLCFA) into cells to be degraded by peroxisomes or lysosomes.

Genetic mutations to ABCD1 result in a deficiency of ALDP, therefore causing a build-up of VLCFAs within the blood and body tissues. The accumulation of VLCFAs is thought to be toxic to myelin in the spinal cord (and brain) as well as the adrenal glands and testes.

Risk Factors of Adrenomyeloneuropathy

Adrenoleukodystrophy is a inherited disease, and someone with a family history of illness is at risk. Because the disorder is passed by parents to their offspring as an X-linked genetic mutation, males are mainly affected, while some females may have signs of the disease but are mostly asymptomatic carriers.

What are the symptoms of AMN?

Like all of the categories of X-ALD, the symptoms of AMN can be quite variable. Below we have listed some of the symptoms that could be present, along with definitions as necessary.

  • Difficulty in walking/change in walking pattern: this is the most common initial symptom of patients with AMN
  • Spastic paraparesis: gradual, progressive weakness and stiffness of the legs; in AMN this is often specific to the lower limbs
  • Ataxia: loss of the ability to coordinate muscle movement
  • Hypertonia: excessive muscle tone
  • Visual defects
  • Dysarthria: Difficulty in articulating words, caused by impairment of the muscles used in speech
  • Seizures: sudden convulsions/attacks/spasms
  • Adrenal insufficiency: The adrenal glands are located above the kidneys, and are responsible for releasing certain hormones such as adrenaline and cortisol. These hormones are important in the control of blood pressure, heart rate, and sexual development and reproduction. In adrenal insufficiency, these hormones are not produced at the appropriate levels and so these processes are not properly controlled. Adrenal insufficiency occurs in approximately 70% of men with AMN.
  • Sexual dysfunction/impotence – This may be related either to involvement of the spinal cord or the testes themselves. The latter is relatively uncommon. It can be diagnosed by measuring testosterone levels in plasma.
  • Bulbar palsy
  • Behavioral changes
  • Bladder dysfunction
  • Mild peripheral neuropathy
  • Weight loss
  • Nausea

Possible Complications

These complications can occur:

  • Adrenal crisis
  • Vegetative state
  • Erectile dysfunction
  • Problems with bowel and bladder control due to sphincter dysfunction
  • Urinary dysfunction
  • Balding and thinning of hair


AMN can be diagnosed by a simple blood test that analyzes the amount of very long chain fatty acids; the levels of these molecules are elevated in X-ALD (see our general fact sheet on X-ALD). A DNA-based blood test is also available.

If the blood test suggests X-ALD, then generally an MRI will be performed in order to assess cerebral involvement. Additionally, the patient will be evaluated for adrenal insufficiency (by another blood test), as this is a common symptom of the disease that can be corrected.

Treatment for Adrenomyeloneuropathy

Currently there is no cure for AMN; however, there are some clinical and dietary treatments that patients are using to help alleviate some of the symptoms of the disease.

One of the possible symptoms of patients with AMN is adrenal insufficiency. The adrenal glands are located above the kidneys, and are responsible for releasing certain hormones such as adrenaline and cortisol. These hormones are important in the control of blood pressure, heart rate, and sexual development and reproduction. In adrenal insufficiency, these hormones are not produced at the appropriate levels and so these processes are not properly controlled. Adrenal insufficiency can be corrected by steroid replacement therapy, which generally will improve the quality of life of the patient. Failure to test for and treat adrenal insufficiency can lead to a fatal outcome. Only replacement dosage of steroids, which do not cause the side effects of “pharmacologic” doses, are required.


The long-term outlook (prognosis) for people with adrenomyeloneuropathy (AMN) varies depending on the subtype. In general, AMN with cerebral involvement (both brain and spinal cord affected) has a worse prognosis than AMN without cerebral involvement (only spinal cord affected). Many people without cerebral involvement are able to maintain successful personal and professional lives with physical therapy, management of bladder control issues, and counseling.

Prevention of Adrenomyeloneuropathy

Genetic counseling is recommended for couples with a family history of X-linked adrenoleukodystrophy. Mothers of affected sons have an 85% chance of being a carrier for this condition.

Prenatal diagnosis of X-linked adrenoleukodystrophy is also available. It is done by testing cells from chorionic villus sampling or amniocentesis. These tests look for either a known genetic change in the family or for very long chain fatty acid levels.

Emphysema is a lung condition that causes shortness of breath. In people with this condition, the air sacs in the lungs (alveoli) are damaged.

What is emphysema?

Emphysema is a lung condition that causes shortness of breath. In people with this condition, the air sacs in the lungs (alveoli) are damaged. Over time, the inner walls of the air sacs weaken and rupture — creating larger air spaces instead of many small ones. This reduces the surface area of the lungs and, in turn, the amount of oxygen that reaches your bloodstream.

When you exhale, the damaged alveoli don’t work properly and old air becomes trapped, leaving no room for fresh, oxygen-rich air to enter. Most people with emphysema also have chronic bronchitis. Chronic bronchitis is inflammation of the tubes that carry air to your lungs (bronchial tubes), which leads to a persistent cough. Emphysema and chronic bronchitis are two conditions that make up chronic obstructive pulmonary disease (COPD). Smoking is the leading cause of COPD. Treatment may slow the progression of COPD, but it can’t reverse the damage.

Types of Emphysema

Emphysema is a type of COPD, and there are different types of emphysema, depending on which part of the lungs it affects.

These are:

  • paraseptal emphysema
  • centrilobular emphysema, which affects mainly the upper lobes and is most common in people who smoke
  • panlobular emphysema, which affects both the paraseptal and centrilobular areas of the lungs

During diagnosis, a CT scan can show which type of emphysema is present. The type does not affect the outlook and treatment.


Emphysema is a pathologic diagnosis defined by permanent enlargement of airspaces distal to the terminal bronchioles. This leads to a dramatic decline in the alveolar surface area available for gas exchange. Furthermore, loss of alveoli leads to airflow limitation by 2 mechanisms. First, loss of the alveolar walls results in a decrease in elastic recoil, which leads to airflow limitation. Second, loss of the alveolar supporting structure leads to airway narrowing, which further limits airflow.

It has 3 morphologic patterns:

  • Centriacinar
  • Panacinar
  • Distal acinar, or paraseptal

Centriacinar emphysema is characterized by focal destruction limited to the respiratory bronchioles and the central portions of the acini. This form of emphysema is associated with cigarette smoking and is typically most severe in the upper lobes.

Panacinar emphysema involves the entire alveolus distal to the terminal bronchiole. The panacinar type is typically most severe in the lower lung zones and generally develops in patients with homozygous alpha1-antitrypsin (AAT) deficiency.

Distal acinar emphysema, or paraseptal emphysema, is the least common form and involves distal airway structures, alveolar ducts, and sacs. This form of emphysema is localized to fibrous septa or to the pleura and leads to formation of bullae (as seen in the images below). The apical bullae may cause pneumothorax. Paraseptal emphysema is not associated with airflow obstruction.

The gradual destruction of alveolar septae (shown in the image below) and of the pulmonary capillary bed in emphysema leads to a decreased ability to oxygenate blood. The body compensates with lowered cardiac output and hyperventilation. This V/Q mismatch results in relatively limited blood flow through a fairly well oxygenated lung with normal blood gases and pressures in the lung, in contrast to the situation in chronic bronchitis. Because of low cardiac output, the rest of the body also suffers from tissue hypoxia and pulmonary cachexia. Eventually, these patients develop muscle wasting and weight loss and are identified as “pink puffers.”


  • The main cause is exposure to airborne irritants, which include tobacco smoke, marijuana smoke, air pollutants, and manufacturing smoke.
  • Very rarely, emphysema is caused by heredity, wherein a person has a shortage of a protein that guards the elastic structure in the lungs, known as alpha-1 antitrypsin deficiency.
  • Close relatives of people with emphysema are more likely to develop the disease themselves. This is probably because the tissue sensitivity or response to smoke and other irritants may be inherited. The role of genetics in the development of emphysema, however, remains unclear.
  • Abnormal airway reactivity, such as bronchial asthma, has been shown to be a risk factor for the development of emphysema.

Who is at risk for emphysema?

The risk factors include

  • This the main risk factor. Up to 75 percent of people who have emphysema smoke or used to smoke.
  • Long-term exposure to other lung irritants, such as secondhand smoke, air pollution, and chemical fumes and dusts from the environment or workplace.
  • Most people who have emphysema are at least 40 years old when their symptoms begin.
  • This includes alpha-1 antitrypsin deficiency, which is a genetic condition. Also, smokers who get emphysema are more likely to get it if they have a family history of COPD.

Symptoms of emphysema

The symptoms include:

  • Breathlessness with exertion, and eventually breathlessness most of the time in advanced disease
  • Susceptibility to chest infections
  • Cough with phlegm production
  • Fatigue
  • Barrel-shaped chest (from expansion of the ribcage in order to accommodate enlarged lungs)
  • Cyanosis (a blue tinge to the skin) due to lack of oxygen.

Complications of Emphysema

People who have emphysema are also more likely to develop:

Collapsed lung (pneumothorax). A collapsed lung can be life-threatening in people who have severe emphysema, because the function of their lungs is already so compromised. This is uncommon but serious when it occurs.

Heart problems. Emphysema can increase the pressure in the arteries that connect the heart and lungs. This can cause a condition called cor pulmonale, in which a section of the heart expands and weakens.

Large holes in the lungs (bullae). Some people with emphysema develop empty spaces in the lungs called bullae. They can be as large as half the lung. In addition to reducing the amount of space available for the lung to expand, giant bullae can increase your risk of pneumothorax.

How do doctors diagnose Emphysema?

Your chest feels tight, you’re out of breath much of the time, and you have a cough that won’t go away. Do you have emphysema? You can’t go on symptoms alone. See your doctor. They’ll do the following tests to find out for sure:

Medical History

Your doctor will talk to you about your health and any recent changes you might have noticed. If you have emphysema, you’ll probably have had shortness of breath, often over a period of months or years. You may also experience wheezing. You might have a cough that won’t go away, too.

Physical Exam

Your doctor will check your weight and blood pressure. They’ll listen to your heartbeat and keep an eye out for anything that seems strange or unusual.

If you have advanced emphysema, your doctor may notice that you have any of the following:

  • You have a “barrel chest” caused by larger-than-normal lungs.
  • You’re wheezing, having a hard time getting air out of your lungs.
  • Your fingertips are rounded. Doctors call this “clubbing.”
  • You purse your lips when you breathe, like you’re blowing a kiss.
  • The oxygen levels in your blood are low (hypoxemia).
  • The carbon dioxide levels in your blood are high (hypercarbia), because emphysema makes it hard to exhale properly.
  • Your lips have a blue tinge (cyanosis), another sign of low oxygen in your blood.
  • Malnutrition causes muscles to slowly waste away in advanced emphysema.

Pulmonary Function Tests (PFTs)

For this exam, you may sit inside an enclosed booth and breathe into a tube. This will allow your doctor to measure:

  • How much air your lungs can hold
  • How fast you can blow air out of your lungs
  • How much air stays trapped in your lungs after you exhale
  • Whether you’re able to breathe better after using medicines you inhale, such as albuterol

If you have normal lungs, you’ll likely be able to empty most of the air from them in 1 second. If you have emphysema, it’ll probably take longer.

Chest X-ray and CT scan

If you have advanced emphysema, your lungs will appear to be much larger than they should be. In early stages of the disease, your chest X-ray may look normal. Your doctor can’t diagnose emphysema with an X-ray alone.

A CT scan of your chest will show if the air sacs (alveoli) in your lungs have been destroyed. These make it hard for you to breathe out like normal.

Arterial blood gas

This test measures the amount of oxygen and carbon dioxide in blood from an artery. It is a test often used as emphysema worsens. It is especially helpful in determining if a patient needs extra oxygen.

Pulse oximetry

This test is also known as an oxygen saturation test. Pulse oximetry is used to measure the oxygen content of the blood. This is done by attaching the monitor to a person’s finger, forehead, or earlobe.

Electrocardiogram (ECG)

ECGs check heart function and are used to rule out heart disease as a cause of shortness of breath.

How is emphysema treated?

There is no cure for emphysema. Treatment aims to reduce symptoms and slow the progression of the disease with medications, therapies, or surgeries.

If you are a smoker, the first step in treating this condition is to quit smoking, either with medications or cold turkey.


Bronchodilator Medications

Inhaled as aerosol sprays or taken orally, bronchodilator medications may help to relieve symptoms of emphysema by relaxing and opening the air passages in the lungs.


Inhaled as an aerosol spray, steroids can help relieve symptoms of emphysema associated with asthma and bronchitis. Over time, however, inhaled steroids can cause side effects, such as weakened bones, high blood pressure, diabetes and cataracts. It is important to discuss these side effects with your doctor before using steroids.


Antibiotics may be used to help fight respiratory infections common in people with emphysema, such as acute bronchitis, pneumonia and the flu.


Patients with emphysema should receive a flu shot annually and pneumonia shot every five to seven years to prevent infections.

Oxygen Therapy

As a patient’s disease progresses, they may find it increasingly difficult to breathe on their own and may require supplemental oxygen. Oxygen comes in various forms and may be delivered with different devices, including those you can use at home.

Surgery or Lung Transplant

Lung transplantation may be an option for some patients with emphysema. For others, lung volume reduction surgery, during which small wedges of damaged lung tissue are removed, may be recommended.

Protein Therapy

Patients with emphysema caused by an alpha-1 antitrypsin (AAT) deficiency may be given infusions of AAT to help slow the progression of lung damage.

Pulmonary Rehabilitation

An important part of emphysema treatment is pulmonary rehabilitation, which includes education, nutrition counseling, learning special breathing techniques, help with quitting smoking and starting an exercise regimen. Because people with it are often physically limited, they may avoid any kind of physical activity. However, regular physical activity can actually improve a patient’s health and wellbeing.

Lifestyle changes

  • Quitting smoking if you are a smoker. This is the most important step you can take to treat this condition.
  • Avoiding secondhand smoke and places where you might breathe in other lung irritants
  • Ask your health care provider for an eating plan that will meet your nutritional needs. Also ask about how much physical activity you can do. Physical activity can strengthen the muscles that help you breathe and improve your overall wellness.

Prevention of Emphysema

To prevent emphysema:

  • If you smoke, talk to your doctor about how to quit
  • Avoid exposure to secondhand smoke
  • Avoid exposure to air pollution or irritants
  • Wear protective gear if you are around irritants or toxins on the job

So sad as three guned down by armed robbery in Delta state.

Gang of robbers have gun down three persons while trying to escape from scene of an operation in Delta State.

The victims, all males, were killed by the robbers who shot indiscriminately while escaping at the Mofor Park in Udu Local Local Government Area of the state at about 10:00 a.m. on Thursday, December 30.

According to an eyewitness, the fleeing robbers shot at the victims while trying to clear their way towards the Otokutu axis of the DSC expressway.

They were coming from Udu and heading towards Otokutu area. So it seems they were stuck at that Mofor Park area and fired the shots, killing the four men.” the witness said.

Spokesperson of the Delta State Police Command, DSP Bright Edafe, who confirmed the incident in a statement on Friday, said two of the robbers were injured during gun duel with police operatives. One of them later died in the hospital while receiving treatment.

The PPRO said the gang’s operational vehicle, one AK 47 Rifle, three locally made guns and eleven rounds of 7.62mm ammunition. 

On 30/12/2021 at about 1005hrs information was received by the Command that dare devil armed hoodlums were firing intense gunshots at Morfor motor garage in Udu LGA and in the process shot three (3) persons dead, Edafe stated.

The Commissioner of Police Delta State CP Ari Muhammed Ali directed the Dpo Ovwian Aladja to go after the hoodlums.

The hoodlums on sighting the police escaped towards Udu-Otokutu expressway in a red mercury mariner jeep with reg no.

Abuja KWL 895 RQ but on getting to Igbeki in Ovwian Aladja they again attempted to attack one chief (name withheld) unknown to them that the chief had a team of Special protection unit (SPU) operatives escorting him.

However, on noticing the presence of the SPU operatives, the hoodlums numbering about four diverted to a street in Igbeki Udu.

The combined team of SPU Operatives and Ovwian Aladja Patrol teams led by the DPO Ovwian Aladja Division went after the hoodlums.

The hoodlums engaged the police teams in a gun duel and in the ensuing shootout, two of the armed robbers sustained gunshot injuries while the other members of the gang escaped into the bush

The injured suspects were taken to the hospital but gave up the ghost while receiving treatment.

One AK 47 rifle with breech no. 148399, one magazine loaded with eleven rounds of 7.62mm live ammunition, three cut-to-size locally made guns were recovered.

One Red Mercury Mariner Jeep with reg no. Abuja KWL 895 RQ which is the operational vehicle of the hoodlums was also recovered. Manhunt for the fleeing suspects is ongoing the PPRO added.


Again the Nigeria government extend NIN validation deadline.

It was learnt that the Nigeria Government has again extend, the deadline for the verification of National Identification Number (NIN) data against Subscriber Identity Module (SIM) cards.

Initially the government had fixed December 31, 2021, as the deadline for the exercise.

During a joint statement signed by Dr. Ike Adinde, Director Public Affairs, Nigerian Communications Commission (NCC) and Mr. Kayode Adegoke, the Head of National Identity Management Commission (NIMC), March 31, 2022, was announced as the new date.

The Honourable Minister of Communications and Digital Economy, Prof. Isa Ali Ibrahim (Pantami) FBCS, FNCS, FIIM, wishes to convey the approval of the Federal Government to further extend the deadline for the National Identification Number (NIN)-Subscriber Identity Module (SIM) data verification to the 31st of March, 2022.