Hashimoto’s Disease or Hashimoto’s Thyroiditis is a condition in which your immune system attacks your thyroid.

Description

Hashimoto’s Disease or Hashimoto’s Thyroiditis is a condition in which your immune system attacks your thyroid, a small gland at the base of your neck below your Adam’s apple. The thyroid gland is part of your endocrine system, which produces hormones that coordinate many of your body’s functions. Inflammation from Hashimoto’s disease, also known as chronic lymphocytic thyroiditis, often leads to an underactive thyroid gland (hypothyroidism). Hashimoto’s disease is the most common cause of hypothyroidism.

It primarily affects middle-aged women but can also occur in men and women of any age and in children. Doctors test your thyroid function to help detect Hashimoto’s disease. Treatment of Hashimoto’s disease with thyroid hormone replacement usually is simple and effective.

Mechanism of Hashimoto’s Thyroiditis

The thyroid gland makes two main hormones – thyroxine (T4) and tri-iodothyronine (T3). Two brain structures, the pituitary gland and the hypothalamus, regulate the hormones released by the thyroid gland. The steps in the process are:

  • The chain of command begins at the hypothalamus, which prompts the pituitary gland to make a chemical called thyroid-stimulating hormone (TSH).
  • The pituitary gland checks the amount of T4 and T3 in the blood and releases TSH if the T4 and T3 levels need to be topped up.
  • \The thyroid gland secretes T4 and T3 depending on the ‘order’ it receives from the pituitary gland. Generally speaking, the more TSH the thyroid receives, the more T4 and T3 it secretes.
  • The pituitary gland may order the thyroid gland to make T4 and T3 but, in the case of Hashimoto’s disease, the thyroid gland can’t deliver.
  • The immune system creates antibodies that attack thyroid tissue. The thyroid gland becomes inflamed (thyroiditis) and thyroid cells become permanently damaged, which hampers the thyroid’s ability to make T4 and T3.
  • In response, the pituitary gland secretes more thyroid-secreting hormone (TSH).
  • The thyroid may enlarge (goitre) as it attempts to obey the pituitary gland.

Causes of Hashimoto’s Thyroiditis

The exact cause of Hashimoto’s is not known, but many factors are believed to play a role. They include:

  • People who get Hashimoto’s often have family members who have thyroid disease or other autoimmune diseases. This suggests a genetic component to the disease.
  • Hashimoto’s affects about seven times as many women as men, suggesting that sex hormones may play a role. Furthermore, some women have thyroid problems during the first year after having a baby. Although the problem usually goes away, as many as 20% of these women develop Hashimoto’s years later.
  • Excessive iodine. Research suggests certain drugs and too much iodine, a trace element required by your body to make thyroid hormones, may trigger thyroid disease in susceptible people.
  • Radiation exposure. Increased cases of thyroid disease have been reported in people exposed to radiation, including the atomic bombs in Japan, the Chernobyl nuclear accident, and radiation treatment for a form of blood cancer called Hodgkin’s disease.

Who is at risk for Hashimoto’s thyroiditis?

Things that may make it more likely to you for to get Hashimoto’s thyroiditis are:

  • Being a woman. Women are about 7 times more likely to have the disease. Hashimoto’s thyroiditis sometimes begins during pregnancy.
  • Middle age. Most cases happen between 40 to 60 years of age. But it has been seen in younger people.
  • The disease tends to run in families. But no gene has been found that carries it.
  • Autoimmune diseases. These health problems raise a person’s risk. Some examples are rheumatoid arthritis and type 1 diabetes. Having this type of thyroiditis puts you at higher risk for other autoimmune illnesses.

What are the symptoms of Hashimoto’s disease?

Some people may not have any symptoms at first. As the disease slowly progresses, the thyroid gland becomes enlarged (a condition called a goiter). A goiter is a common first sign of Hashimoto’s disease. A goiter is painless, but can create a feeling of fullness in the throat, and can make the front of your neck look swollen.

Other symptoms of Hashimoto’s disease that develop over time include:

  • Tiredness (fatigue)
  • Weight gain
  • Feeling cold
  • Joint stiffness and muscle pain
  • Constipation (trouble having a bowel movement)
  • Depression
  • Puffy eyes/face
  • Dry skin
  • Thinning hair/hair loss
  • Heavy or irregular periods
  • Difficulty becoming pregnant
  • Memory problems/difficulty thinking or concentrating
  • Slow heartbeat

Complications related to Hashimoto’s Thyroiditis

If left untreated, Hashimoto’s thyroiditis can cause complications, some of which can be severe. These can include:

  • Heart problems, including heart failure
  • Anemia
  • Confusion and loss of consciousness
  • High cholesterol
  • Decreased libido
  • Depression
  • Myxedema
  • Birth defects
  • Goiter

Hashimoto’s can also cause problems during pregnancy. Recent researchTrusted Source suggests that women with this condition are more likely to give birth to babies with heart, brain, and kidney defects. In order to limit these complications, it’s important to monitor thyroid function over the course of pregnancy in women who have thyroid problems.

How is Hashimoto disease diagnosed?

Your doctor will perform a physical examination and order blood tests to measure your hormone levels; we collectively call these labs thyroid function tests (TFTs) – TSH, free T4, and total T3.

  • TSH test: TSH is hormone released from your pituitary gland when there is not enough thyroid hormone in the system. TSH will be high if there is not enough thyroid hormone in the system. Normal ranges for TSH vary lab by lab and also are dependent on age (TSH normally rises as we age).
  • Free T4 and total T3 test: T3 and T4 are thyroid hormones. A low level of free T4 and/or T3 test also suggest hypothyroidism
  • Thyroid peroxidase (anti-TPO) antibody blood test: This test detects the presence of antibodies directed against the thyroid. Most people with Hashimoto disease have these antibodies, but people whose hypothyroidism is caused by other conditions may not. TPO antibodies may also be present in normal people without hypothyroidism.

Treatment for Hashimoto’s Disease or Hashimoto’s Thyroiditis

Depending on the patient’s condition, treatment comprises of observation and medications. If the patient is not suffering from any hormone deficiency and if the thyroid function is normal, then there is no need for medications right away and your physician may propose a wait-and-see approach. If the thyroid is not functioning normally; and if the patient has symptoms, then medications are started and more often than not, they are needed to be taken for lifetime. The following medications are given:

Synthetic Hormones

As there is deficiency of the thyroid hormone, replacement therapy is done with synthetic thyroid hormone, such as levothyroxine (Synthroid, Levoxyl etc.) which is a synthetic thyroid hormone. Synthetic levothyroxine is same as thyroxine, which is naturally produced by the thyroid gland. Synthetic levothyroxine helps in restoring normal hormone levels and reverses or negates the hypothyroidism symptoms.

Dosage Monitoring

Depending on the TSH level, which should be checked every six to 12 months, the dosage needs to be changed accordingly. It is very important to monitor the dosage of levothyroxine. To do this, your physician will be checking your TSH level within a few weeks of starting the treatment, as excessive thyroid hormone causes increase in bone loss, resulting in increased risk of osteoporosis. Overdose of levothyroxine can also lead to arrhythmias (heart rhythm disorders).

In patients with CAD or coronary artery disease, the doctor will start the treatment with a low dose of the medication and slowly increase the dose, so that the heart can adapt to the metabolism. When used in the right amount of dosage, there are literally no side effects from levothyroxine. It is important to continue with the same brand and let the doctor know if you have changed the brand. It is also important to take the dose regularly and not miss any doses or abruptly stop the medicine, as this will result in recurrence of all the signs and symptoms of this disease.

Drug Interaction

There are some supplements, medications and food groups, which can affect the way levothyroxine is absorbed. To avoid this, levothyroxine should be taken at least a minimum of four hours after or before taking other medications. It’s important to let your doctor know if you are taking any huge amounts of a high-fiber diet or soy products or any of the following things:

  • Supplements, such as calcium and iron; and multivitamins containing iron.
  • Medication for lowering the level of blood cholesterol, such as Prevalite (cholestyramine).
  • Antacids which contain aluminum hydroxide.
  • A drug used for preventing high blood potassium levels, i.e. sodium polystyrene sulfonate.
  • Sucralfate, which is medication given for treatment of ulcers.

Prevention of Hashimoto’s Thyroiditis

Unfortunately, there is no known way to prevent Hashimoto’s thyroiditis (or inflammation of the thyroid gland. But on the bright side, this disorder is very treatable. The sooner you get diagnosed, the sooner you can start receiving treatment.

Macular edema or Diabetic Macular Edema, is swelling or thickening of the eye’s macula, the part of your eye responsible for detailed, central vision.

Definition

Macular edema or Diabetic Macular Edema, is swelling or thickening of the eye’s macula, the part of your eye responsible for detailed, central vision. The macula is a very small area at the center of the retina a thin layer of light-sensitive tissue that lines the back of the eye. Light rays are focused onto the retina, where they are transmitted to the brain and interpreted as the images you see. It is the macula that is responsible for your pinpoint vision, allowing you to read, sew or recognize a face.

Macular edema develops when blood vessels in the retina are leaking fluids. The macula does not function properly when it is swollen. Vision loss may be mild to severe, but in many cases, your peripheral (side) vision remains. Macular edema is often a complication of diabetic retinopathy, and is the most common form of vision loss for people with diabetes particularly if it is left untreated.

Epidemiology

Diabetes is the leading cause of new blindness in the United States, and clinically significant macular edema (CSME) contributes greatly to this vision loss. In the absence of ophthalmologic treatment, persons with diabetes have a 25-30% risk of moderate vision loss. With treatment, the risk drops by 50%. According to 2007 data, 23.6 million people in the United States have diabetes, but only 17.9 million have been diagnosed. About 50% of those with diagnosed diabetes do not receive appropriate eye care. The World Health Organization estimates that worldwide, more than 150 million people have diabetes.

Although diabetes is more common in Hispanics, African Americans, and Native Americans than in whites, no data describe a greater risk of developing macular edema among diabetic patients of any one racial group. Likewise, no data describe a difference in risk of diabetic macular edema between the sexes.

Types of Macular Edema

It can be broadly characterized into two main anatomic categories:

  • Focal Macular Edema is characterized by specific areas of separate and distinct leakage in the macula with sufficient macular blood flow.
  • Diffuse Macular Edema results from leakage of the entire capillary bed surrounding the macula, resulting from a breakdown of the inner blood-retina barrier of the eye.

In addition to Focal and Diffuse, DME is also categorized based on clinical exam findings into clinically significant macular edema (CSME), non-CSME and CSME with central involvement (CSME-CI), which involves the fovea.

Macular Edema Risk factors

Anyone with type 1 or type 2 diabetes is at risk for developing diabetic macular edema (DME) or diabetic retinopathy. Specific risk factors include:

  • Duration of diabetes – the longer you have diabetes, the greater your risk of developing diabetic macular edema (DME)
  • Chronic high levels of blood sugars
  • High cholesterol levels
  • Hypertension (high blood pressure)
  • Kidney disease
  • Heart disease
  • Smoking
  • Pregnancy

Causes of Macular Edema

Usually the cause is that tiny retina capillaries leak from weak vessel walls into the center of the macula. The patient perceives fogginess in the middle or just to the side of the central visual field, The loss of vision can occur slowly. It rarely causes an enduring loss of vision, but can take 2 to 15 months to heal. Sometimes macular swelling can occur after cataract or other surgery but is, with treatment, usually of temporary duration.

Because many tiny capillaries surround the macula, any condition affecting blood circulation in the body (diabetes, atherosclerosis, etc.) affects the eye; the result may be macular edema. The reason this is important is that macular edema is usually a symptom of a more serious health problem that needs to be addressed. In the case of the latter, macular edema typically occurs within 1-2 months post-surgery; though it can even happen many months, and even years, afterward.

Here is a list of the possible key causes of macula edema:

Oxidative stress: The imbalance between the antioxidant defense system, production of free radicals (reactive oxygen species, ROS), cause oxidative stress in the body and in the tissues of the eye. Damage from ROS causes a host of problems, including inflammation and fluid leakage in the macula and other parts of the eye.

Other eye conditions: Macular edema is caused by many diseases, including optic neuritis, retinal vein occlusion, macular degeneration, diabetic macular leakage, eye inflammation, idiopathic central serous chorioretinopathy, anterior or posterior uveitis, pars planitis, retinitis pigmentosa, radiation retinopathy, posterior vitreous detachment, epiretinal membrane formation, idiopathic juxtafoveal retinal telangiectasia, capsulotomy or iridotomy. These disorders can be due to circulatory problems associated with hypertension, high cholesterol, etc.

Other health conditions: The disorder can be related to general circulatory problems linked to hypertension, high cholesterol, and to nervous system-related conditions such as multiple sclerosis.

Diabetes: A 2006 study of diabetics determined that 9% had macular edema; more recent evaluations find 3.8% in diabetics over 40. High levels of hemoglobin A1c, a biomarker for diabetes, are associated with higher diabetic macular edema risk. Having diabetes for 10 years or longer increases the risk.

Medicines: A history of use of topical epinephrine or prostaglandin analogs for glaucoma is a factor.

Eye surgery: Surgery for cataracts, glaucoma, or other retinal disease can result in macular edema, sometimes months after the surgery. Edema can occur due to post-operative lesions.

Ethnicity: Non-Hispanic Blacks are at greater risk of developing diabetic macular edema.

Injury: If you injure your eye and notice fuzziness in your central vision, be sure to see your eye doctor immediately.

Inflammation: Inflammatory disease disorders such as cytomegalovirus infection, retinal necrosis, sarcoidosis, Behçet’s syndrome, toxoplasmosis, Eales’ disease, and Vogt-Koyanagi-Harada syndrome are often accompanied by macular edema.

Drugs: History of use of topical epinephrine or prostaglandin drugs for glaucoma.

Surgery: Cataract or other eye surgery

Men are at higher risk

Symptoms of Macular Edema

The symptoms will vary according to how swollen the macula is and whether it is in one or both eyes.

Most people will notice one or more of the following:

  • Blurred or wobbly vision
  • Vision loss/difficulty reading
  • Washed-out colours

If you are struggling with any of these symptoms, see an optometrist as soon as possible. Severe and untreated macular oedema can result in long lasting and severe damage to your eyesight.

Macular Edema Complications

Serious Complications Associated With Macular Edema are as follows:

  • Untreated chronic macular edema can lead to permanent damage to the retinal architecture and permanent vision loss.
  • Irvine-Gass syndrome, also known as postoperative macular edema, is a common complication of cataract surgery.
  • A prior history of diabetes, retinal detachment, ocular inflammation, retinal vein occlusion, epiretinal membrane, and ocular prostaglandin use have all been associated with an increased risk of macular edema after cataract surgery.
  • In addition, intraoperative complications during cataract surgery have been associated with a higher incidence of postoperative macular edema.
  • The pathogenesis of macular edema after cataract surgery is thought to be multifactorial, but the core mechanism is likely surgically induced inflammation.
  • Intraocular inflammation upregulates endogenous inflammatory mediators that increase vasopermeability, leading to the development of macular edema.

Diagnosis and test

To diagnose macular edema, your eye care professional will conduct a thorough eye exam and look for abnormalities in the retina. The following tests may be done to determine the location and extent of the disease:

Visual acuity test: A visual acuity test is a common way to identify vision loss and can help to diagnose vision loss as a result of macular edema. This test uses a standardized chart or card with rows of letters that decrease in size from top to bottom. Covering one eye, you will be asked to read out loud the smallest line of letters that you can see. When done, you will test the other eye.

Dilated eye exam: A dilated eye exam is used to more thoroughly examine the retina. It gives additional information about the condition of the macula and helps detect the presence of blood vessel leakage or cysts. Drops are placed in your eyes to widen, or dilate, your pupils. Your eye care professional then examines your retina for signs of damage or disease.

Fluorescein angiogram: If earlier tests indicate you could have macular edema, your eye care professional may perform a fluorescein angiogram. In this test, a special dye is injected into your arm and a camera takes photos of the retina as the dye travels through the blood vessels. This test helps your ophthalmologist identify the amount of damage to the macula.

Optical coherence tomography: This is a test that uses a special light and a camera for detailed views of the cell layers inside the retina. It detects the thickness of the retina and so it’s useful in determining the amount of swelling in the macula. Your eye care professional may also use optical coherence tomography after your treatment to track how well you are healing.

The Amsler Grid: The Amsler Grid provides an easy way to test whether or not your central vision has changed. It can recognize even small changes in your vision.

The Amsler Grid

If you need reading glasses, wear them when you look at the Amsler grid. The grid should be at the same distance from your eyes as your usual reading material about 14 inches. Test both eyes, one at a time, to see if any parts of the grid look distorted, missing, or dark. Mark the areas of the chart that you’re not seeing properly and bring it with you to your next eye exam.

Macular edema in eye

Treatment and medications

The necessary treatment for macular edema varies for each patient depending on the severity of vision loss, underlying retinal conditions, and how early this condition has been identified.

Patients that have been diagnosed with diabetic retinopathy will notice that there is overlap in treatments. Please click upon the name of the treatment to be redirected to a page with a more detailed description.

Focal Macular Laser Treatment: This laser treatment is used to control bleeding and leakage caused by abnormal blood vessels. During laser treatment, a series of small, painless burns are made to the abnormal blood vessels. This seals the leaks and prevents further bleeding into the retinal.

Avastin or Lucentis Injections: Avastin and Lucentis are both anti-VEGF medications that are injected into the eye. These medications block the chemical responsible for the leakage. Avastin and Lucentis help mitigate the vision loss caused by fluids leaking from these blood vessels.

Periocular and Intravitreal Steroid Injections: Periocular and intravitreal injections of corticosteroids, such as Kenalog, can be used to manage macular edema in conjunction with laser treatment. Regular injections of steroids help to reduce swelling of the macula and leakage of abnormal blood vessels.

Prevention of Macular Edema

  • Maintaining good blood sugar, blood pressure, and cholesterol control helps prevent DME.
  • Receiving a comprehensive dilated eye exam at least once a year, or more often as directed by the eye doctor.
  • Pregnant woman with diabetes should have a comprehensive dilated eye exam as soon as possible. The eye doctor may recommend additional exams during pregnancy.
  • Maintaining a healthy lifestyle, exercising regularly, and having a healthy diet. Talk to a doctor before starting an exercise program.

Anosmia is the full loss of smell. Anosmia can be a temporary or permanent condition.

Definition

Anosmia is the full loss of smell. Anosmia can be a temporary or permanent condition. You can partially or completely lose your sense of smell when the mucus membranes in your nose are irritated or obstructed such as when you have a severe cold or a sinus infection, for example. But if the inability to smell isn’t related to a cold or sinus infection, or it doesn’t return after congestion clears, you should see a doctor. It could be a symptom of another issue.

The sense of smell is important to overall health and nutrition since diminished sensations can lead to poor appetite and malnutrition, especially in the elderly. An altered sense of smell may pose other health-related problems. People with anosmia may accidentally consume soured or rancid foods because they are unable to detect odors that signal spoilage. Those with anosmia may also be unaware when they are breathing toxic, polluted, or smoke-filled air.

Although rare, some people are born without a sense of smell, which is a condition called congenital anosmia. This occurs when there is either an inherited genetic disorder or abnormal development of the olfactory system (the body’s sensory system for smell) occurring before birth. Unfortunately, there is no cure for congenital anosmia.

Types of Anosmia

There are three types of Anosmia:

Temporary: Temporary anosmia is common as it is caused due to a congested nasal pathway that we go through during cold, flu, sinus, etc.

Permanent: This anosmia causes permanent smell loss due to various reasons such as accident, advancing age, etc.

Congenital: This anosmia happens in one out of 10,000 adults where they are born with this condition.

Anosmia risk factors

Different factors that are known to interfere with the smell sense include:

Chemicals– A wide range of industrial chemicals, including heavy metals, inorganic and organic compounds, acids, and pollutants.

Diseases of the hormonal system– Such as diabetes, Cushing’s syndrome, and hypothyroidism.

Diseases of the nervous system– Such as Alzheimer’s disease, multiple sclerosis, migraine, Korsakoff syndrome, brain tumors, brain lesions, and epilepsy.

Drugs– Stimulants (such as amphetamines and cocaine), depressants (such as morphine), some antibiotics, and other drugs, including the vasoconstrictors in nasal sprays.

General diseases– Such as bronchial asthma, leprosy, and cystic fibrosis.

Trauma- Including blows to the head or injuries to the nose.

Causes of Anosmia

Some common causes of anosmia include:

Chronic nasal-sinus disease, e.g., rhinitis (inflammation in the nose caused by viral, bacterial, or fungal infections), nasal polyps, and/or chronic sinus infections can block the passage of odor molecules to the smell receptors or interfere with how the receptors detect odors.

Viral respiratory infections (common colds) – Can damage or destroy the smell receptor cells.

Head injury– Can sever the olfactory nerve fibers that send smell information to the brain and/or damage parts of the brain that process smell information

Aging– Repeated exposure of the olfactory receptors to environmental airborne toxins can damage the smell receptors and/or the receptor cells can lose the ability to regenerate

Congenital or in-born causes – The reasons for this are unknown at this time.

Anosmia Symptoms

Anosmia and taste are linked. Many individuals that lose their sense of smell, or those who are born without a sense of smell, also lose their ability to taste. Scientists believe this is due to the fact that people “taste” food by the smell, texture, and temperature. In addition, many people are able to recognize their foods or beverages by smell. Coffee is a beverage that is not only enjoyed by taste, but also by smell.

If you have many symptoms and signs that accompany your anosmia, you should consider an evaluation from nose and mouth specialist. Symptoms to be concerned about include:

  • Inability to smell
  • Inability to taste
  • Stuffy nose and congestion
  • Persistent sneezing, nasal drainage, and itchy watery eyes
  • Weight loss
  • Headaches
  • Nose Bleeds

Complications of Anosmia

Complications you may experience if you lose your sense of smell:

  • An inability to taste the food, which can lead to eating too much or too little
  • An inability to smell spoiled food, which can lead to food poisoning
  • The increased danger in the event of a fire if you cannot smell smoke
  • Losing the ability to recall smell-related memories
  • Loss of intimacy due to the inability to smell perfume or pheromones
  • Losing the ability to detect chemicals or other dangerous odors in your home
  • Lack of empathy from family, friends, or doctors
  • Inability to detect body odors
  • Mood disorders such as depression
  • Lack of interest in social situations, which might include being unable to enjoy the food at a social gathering

Diagnosis and test

The loss of smell is difficult to measure. Your doctor may ask you some questions about your current symptoms, examine your nose, perform a complete physical examination, and ask about your health history.

The diagnosis is made clinically from the history of a poor sense of smell along with a clinical exam of the nasal passage.

Physical examination: The nasal cavity is examined thoroughly to look at the structure and identify the presence of inflammation or blockage. The sense of smell is examined by asking the individual to identify several common odors while blindfolded.

Laboratory investigations: blood tests to look for vitamin deficiencies, blood sugar levels are performed to screen for illness

Imaging: Scans of the head and brain using CT or MRI are performed to look for any structural defects, tumors or neurological disease and trauma.

Treatment and medications

Fifty percent of anosmia cases can be treated and symptoms reversed, depending on the underlying cause. And in cases that cannot be reversed, symptoms can often be reduced with treatment.

Some anosmia treatments that can help if your loss of smell is from obstruction:

  • A decongestant
  • An antihistamine
  • Steroidal nasal spray
  • Sinus surgery for nasal obstruction, chronic sinusitis or nasal polyps
  • Short term course of oral steroids

More recent innovative therapies that have shown promise in controlled studies in improving the sense of smell include:

  • Intranasal theophylline spray
  • Smell therapy.

Both are offered as part of a comprehensive therapeutic regimen.

Prevention of Anosmia

Avoiding certain chemicals, certain drugs, and not smoking might help prevent you from losing your sense of smell.

Also, since anosmia can be one result of sustaining a brain injury, it’s yet one more reason to be extra careful when playing dangerous sports, driving, or taking part in similar risky activities.

If you do have anosmia, you can make your environment safer by:

  • Making sure your smoke alarms are working
  • Checking that cookers, barbecues, and electrical appliances are turned off properly
  • Reading food expiry dates carefully

Senator Shehu Sani recall that the presidency once accused Saraki of budget padding.

Oneworld learnt that Senator Shehu Sani from Kaduna central was on his Twitter account this afternoon recall about budget padding.

In his tweet, Shehu Sani stated that the presidency once accused the Bukola Saraki led National assembly of budget padding, Shehu Sani said they now have grand Budget padding under Lawan.

Budget padding is define as a way of inflating proposed budget to either expand projects or cover unexpected costs that may arise from the execution of projects.

Sen. Bukola Saraki was Nigeria Senate president from 2015 to 2019.

During that period the National assembly was accused of padding some of the budget passed.

For instance, in the 2017 budget, President Buhari proposed a budget of N7.28 trillion, but the National assembly passed a budget of N7.44 trillion.

This has also happen with the 2022 budget, because president Buhari has expressed worries over some of the changes made to the budget by the National assembly.

Oneworldvisionnews

So Painful. No fewer than 8 persons have been reportedly killed following an accident that occurs along Abakaliki – Enugu road.

Many persons are feared dead on Saturday morning following an accident that occurs along Abakaliki-Enugu expressway around Nkalagu in Ishielu Local Government Area of the State.

The accident, which caused heavy traffic jam along the federal highway, was reported to have led to the death of over 8 persons in the area.

An unconfirmed report has it that all the passengers in the Sienna bus lost their lives following the accident.

The development led to other vehicles parking along the highway as passengers were seen congregating at the scenes of the accident, in order to get more information about the incident.

Commuters and passengers were seen at the scene of the accident discussing about the ugly new year accident.

As at the time of this report, no Federal Road Safety Corps, FRSC, personnel had arrived the location of the accident.

Another accident also occurred at Izhiangbo area of Ohaukwu Local Government Area of the State.

A truck loaded with food stuff fell and blocked a bridge linking the Abakaliki-Enugu expressway.

An FRSC personnel that was at the scene of the accident confirmed to newsmen that no life was lost.

The development, however made motorists to follow alternative route that is close to the bridge to get to their various destinations.

The accident also hindered vehicular movement within the area.

The new year accidents that occurred along Abakaliki/Enugu expressway have send shivers down the spins of road users as many are still wondering what 2022 has in stock for them.

Oneworldvisionnews

Man United defeat Burnley 3-1 at old Trafford.

According to our sports news line it was reported that Manchester United thrashed Burnley 3-1 at Old Trafford, claiming their 9th Premier League victory of the season and securing three crucial points in the race for the top four of the tournament.

The Red Devils goals were scored by Scott McTominay in the 8th minute after clever assist from Cristiano Ronaldo before Jadon Sancho strong effort was deflected in by Ben Mee and Cristiano Ronaldo scored 3rd goal in the 35tg minute.

Burnley scored a consolation goal through Aaron Lennon in the 38th minute to deny the hosts a clean sheet victory at home. 

In the second half, Ralf Rangnick substituted Fred, Dalot, and Varane for Ronaldo, Greenwood, and the injured Baily, and the Red Devils were able to force a 3-1 victory.

Man United moved up to sixth place in the EPL table after the 3-1 victory, with 31 points, overtaking Tottenham Hotspurs, while Burnley dropped to 18th place with 11 points.

Arsenal are currently in fourth place in the EPL table, with 35 points, ahead of their match against Manchester City on Saturday at Emirates Stadium, while second-placed Chelsea will face third-placed Liverpool on Sunday.

Oneworldvisionnews

Ukraine and Georgia excempted from president Putin new year congratulatory message.

Information reaching us reveals that President Vladimir Putin of Russia; Putin did not congratulate the presidents of Ukraine and Georgia on the New Year

In his congratulations remark to US President Joe Biden, Putin wrote that the two countries can and should interact constructively to overcome common challenges.

The Russian President Vladimir Putin have congratulated the heads of 42 states, including the United States and the Vatican.

The congratulations did not include the names of the presidents of Ukraine, Volodymyr Zelensky and Georgia, Salome Zurabishvili. As it was published on the Kremlin website.

President Putin also congratulated Alexander Lukashenko, Chinese President Xi Jinping, German Chancellor Olaf Scholz, British Prime Minister Boris Johnson and Queen Elizabeth II, French President Emmanuel Macron, Turkish President Recep Erdogan, Kazakh President Kassym-Zhomart Tokayev and the country’s first President Nursultan Nazarbayev.

In his congratulations to US President Joe Biden, Putin wrote that the two countries can and should interact constructively to overcome common challenges. The leaders of the Baltic countries Lithuania, Latvia and Estonia were without Putin’s congratulations.

Last year, the Russian president also did not congratulate Zelensky and Zurabishvili on the New Year. In 2019, Putin for the first time in six years congratulated the President of Ukraine on the New Year during a telephone conversation.

Then they discussed the exchange of detained citizens, which took place on December 29, and made lists for future exchanges.

The former Ukraine president, Petro Poroshenko, was never congratulated by Putin.

Record shows that the last time the Russian leader congratulated the Ukrainian was in 2013, before the annexation of Crimea and the deterioration of relations between the countries.

Oneworldvisionnews

New year tragedy: No fewer than Twelve persons have been reportedly abducted by bandits in Kaduna state.

One world learnt that no fewer than 12 persons, including 10 women were kidnapped by bandits in a fresh attack at Kerawa village in Giwa Local Government Area of Kaduna State in the late hours of Friday.

Jamil Kerawa community youth leader, confirmed the incident, and he added that the bandits operated unupposed.

Total 12 persons were abducted by bandits, two male and 10 women, and one person killed in the operation. One other was injured and rushed to hospital in Zaria. We need help in Kerawa because only God is protecting us, He lamented.

Mr. Jamil appealed to the State and federal Government to deploy security personnel in the area, lament about the increased attacks by bandits.

It was also learnt that the neighboring villages like Hayin Dam, Rago, Unguwar Salahu and Zariyawa were also invaded by monster bandits recently.

One Mr. Jafaru a residents said his biological brother and his uncle son were among those abducted.

No official reaction from the state government and Police but the spokesperson of the Nigeria State Police Command has promised to report back after investigation.

Oneworldvisionnews

So Sad as unknown gunmen shot dead a Pastor wife and Kidnapp the Pastor.

It was reported that the wife of the General Overseer of Faith Fasting and Prayer Ministry in Abraka, Ethiope East Local Government Area of Delta State, Pastor (Mrs) Faith, have been killed by unknown gunmen, and her husband, Prophet Jay Jay Enejeta kidnapped, during the operation.

According an eyewitness a Pastor and his wife was attacked on Thursday evening on their way back home from evening program.

Prophet Enejeta was driving home with his wife when the gunmen opened fire on their Lexus Sports Utility Vehicle (SUV) while approaching their residence.

The gun bullet from the gunmen pierced through the window of the front passenger seat hit the wife in her neck and head, killing her on the spot.

Thereafter, the husband was kidnapped by the unknown gunmen to an unknown place.

Mrs Enejeta was confirmed dead at a hospital she was speed to in Abraka and her remains was deposited.

The Nigeria Police Public Relations Officer (PPRO), Bright Edafe DSP, is yet to reply to the messages of our news source as of the moment of report.

More details coming as soon as possible, Oneworldvisionnews..