Health benefits Of Hot Water In The Body When Taken Regularly?

Do you know that there are certain things that may happen to you if you often drink hot water? Hot water is often seen as one fluid that can help relief some health issues and also provide the body with numerous benefits but some people are unaware of some of the benefits they can obtain from drinking hot water regularly.

So in this article in line with a publication on Medical News Today, we are going to have a look at things that may happen to the body if a person drinks hot water regularly. Just sit tight and enjoy this article while learning something new.

  • Reduced Pain – drinking hot water can improve blood circulation and also boost blood flow to and fro all the parts of the body including injured muscles. Some research has shown that drinking hot water may provide some internal pain relief in the body, but in some cases too may worsen swelling.
  • Body Detoxification – natural health advocates believe that drinking hot water may help the body detoxify because when water is hot enough to raise a person body temperature, it can cause sweating and sweating can help remove toxins from the body and clean a person’s pores. This is one of the effects taking hot water regularly may provide the body.
  • Improves blood circulation – hot water often serves as a vasodilator, which means that it dilates blood vessels improving blood circulation. This dilation of blood vessels can improve blood circulation and help relax and reduce pain in the body.
  • Weight loss – drinking hot water may also reduce weight. The reason is that, drinking more water generally can cause a person to eat less food and when a person eats less food, he or she is automatically set for less weight or weight loss in most cases.

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Nigeria troops rescue over 500 women and children in Sambisa forest.

Nigerian Army Special Forces of Operation Hadin Kai have killed scores of Boko Haram and the Islamic States West Africa Province (ISWAP) terrorists following the storming of 13 camps of the terrorists in Sambisa Forest in the North East of Borno State.

Counter Insurgency Expert in Lake Chad , Zagazola Makama while confirming the offensive the troops successfully cleared the terrorists adding that, The terrorists left behind over 500 women and children including their families and those that they forcefully abducted.


The source said that one of the rescued victims included, Hajja Fusambe, a mother of the popular dreaded Boko Haram Commander known as Alhaji Ari Hajja Fusambe, who was taken to Sambisa forest by her son in 2014.

The Sources added that 50 more trapped women and children surrendered in the early hours of Wednesday to troops of 21 Special Forces Brigade Bama, where they will be profiled and undergo rehabilitation.


Meanwhile, in continuation of the offensive, troops also neutralized several ISWAP/BokoHaram terrorists including the Amir & spiritual head of Galta during an aggressive clearance operation around Manjo Ali Qere in Borno State on Thursday 28 April 2022.

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So painful as popular Nigeria movie actress Chinedu Bernard passes on.

Actress Chinedu Bernard has slumped in a church and confirmed dead in a hospital.

The ‘Big Mama’s Stick’ actress was reportedly confirmed dead by doctors at the East Side hospital in Enugu State.

She was said to have slumped while cleaning the Chapel of St. Leo the Great Catholic Church inside Federal Housing area in Enugu State.

It was learnt one of the Reverend Fathers rushed her to the hospital with some members of the church.

Reports identified the Reverend Father, who assisted in taking the late actress to the hospital as Rev Fr Uchendu Chukwuma.

But the ‘Last Manhood’ actress was declared dead by the doctors.

The cause of the late Bernard’s death remains unknown.

The late Bernard’s remains have been deposited in the morgue.

Fondly referred to as ‘Choco,’ the late Bernard featured in movies such as The Big Mama’s Stick, Money fever, The Mad, The Last Manhood, Mad Love etc.

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(PDP) has so far screened former Vice President Atiku Abubakar, former Senate Presidents Anyim Pius Anyim and Bukola Saraki.

The presidential screening committee of the Peoples Democratic Party (PDP) has so far screened former Vice President Atiku Abubakar, former Senate Presidents Anyim Pius Anyim and Bukola Saraki, who have appeared for the exercise on Friday morning.

The screening exercise, which began at about 11:00am, saw aspirants vying for the PDP presidential ticket appeared at the Legacy House, Abuja, the alternate national zecretariat of the opposition party, where the exercise is currently taking place.

All three aspirants screened so far declined to disclose the outcome of the exercise as it affects their aspiration.

There are 17 presidential aspirants and the process is still on will 14 others to follow.

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The Polish government have successfully sent two hundred tanks to Ukraine.

A massive 200 tanks have been sent to Ukraine by its neighbour, Poland, according to Dailymail.

The heavy weapons were sent to help Ukraine win the war against Russia.

Warsaw sent more than 200 T-72s originally produced by the Soviet Union – into Ukraine in recent weeks, the country’s national radio broadcaster said today, along with mobile artillery, drones and rocket launchers as part of a $1.6bn package.

Ukraine’s allies have massively stepped up support for Kyiv as fierce fighting rages in the east, with Kyiv’s generals today saying that Russia is suffering ‘colossal’ casualties in the pivotal battle for Donbas.

Oleksiy Arestovych, a Ukrainian secret service veteran-turned presidential adviser, acknowledged his military is suffering ‘serious losses’ on battlefields in the east but insisted that Russia’s casualties are ‘much, much worse’.

Meanwhile Ukraine continues to carry out attacks behind Russian lines to cut off vital supply routes, with a fuel dump in the Donetsk region catching fire today.

Kyiv has not acknowledged carrying out any of the attacks – which have also hit railway bridges and ammo dumps – but is widely thought to be orchestrating them.

Earlier this week, a Ukrainian MP described the blasts as ‘karma’.

Russia is pouring troops into the battle in Donbass in an effort to force a bloody victory having been defeated in its initial aim to storm in Kyiv, topple the government, and install a puppet regime loyal to Moscow.

After it became apparent they did not have sufficient force to take the capital, Russia’s generals yanked their units out, patched them up as best they could, and then threw them back into the fight in Donbas.

They also adapted their tactics – abandoning precision missile strikes and rapid advances which saw them mauled around Kyiv in favour of slow advances behind walls of blanket artillery in similar tactics to WW1 trench warfare.

The move has met with mixed success. Ukraine has acknowledged losing control of some towns and villages, but has made gains elsewhere in counter-attacks.

Supplies of heavier weapons including tanks, artillery cannons, precision munitions and anti-aircraft weapons are designed to help with those attacks while ensuring the Ukrainians can destroy as much Russian equipment as possible in the process.   

As evidence of the West’s dedication to the mission, Joe Biden last night pledged $33billion in aid for Ukraine including $20billion in military aid. 

We need this bill to support Ukraine in its fight for freedom, Biden said ‘The cost of this fight it’s not cheap but caving to aggression is going to be more costly.

Zelenskiy tweeted: ‘Thank you @POTUS and the American people for their leadership in supporting Ukraine in our fight against Russian aggression. 

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Terrorist kill two locals and a soldier in Plateau state.

The Special Task Force, Operation Safe Haven (OPSH), maintaining peace in Plateau and environs, says gunmen have killed two locals and a soldier at Rikwe-Chongu village of Kwall District of Bassa Local Government Area of Plateau.

Spokesman for the task force, Major Ishaku Takwa, confirmed the incident on Friday in Jos.

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Outrage as heavily armed men attack bullion van in Imo State.

Gunmen believed to be armed robbers have attacked a bullion van in Imo State, police authorities have said.

The Public Relations Officer of the Imo State Police Command, Mike Abattam, confirmed the attack on Thursday in Owerri, the state capital.

He explained that the incident occurred in the afternoon at Nguru community in Mbaise Local Government Area of the state.

Recounting how the incident happened, Abattam explained that the bullion van was returning to Owerri after delivering money at one of the commercial banks in the local government.

He added that the bullion van was attacked by the gunmen who caught up with the vehicle with the intention to cart away the money it was conveying.

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Russia fires ballistic missile in Kyiv amidst UN Secretary Antonio Guterres visit.

Russian strikes slammed into Kyiv on Thursday evening as UN Secretary-General Antonio Guterres was visiting, in the first such bombardment of Ukraine’s capital since mid-April, the mayor said.

There was no immediate information about casualties from the strikes, which hit a western part of the city centre, but a close aide to the UN chief sent a message to journalists confirming they were safe.

Kyiv mayor Vitali Klitschko said there had been ‘two hits in the Shevchenkovsky district’ in a post on Telegram and said they were clarifying details about casualties.

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Ehlers Danlos syndrome (EDS) is a disease that weakens the connective tissues of your body.

Overview – Ehlers Danlos Syndrome

Ehlers Danlos syndrome (EDS) is a disease that weakens the connective tissues of your body. These are things like tendons and ligaments that hold parts of your body together. EDS can make your joints loose and your skin thin and easily bruised. It also can weaken blood vessels and organs.

It is an inherited condition that affects the connective tissues in the body. Connective tissue is responsible for supporting and structuring the skin, blood vessels, bones, and organs. It’s made up of cells, fibrous material, and a protein called collagen. A group of genetic disorders causes Ehlers-Danlos syndrome, which results in a defect in collagen production.

Types of Ehlers Danlos Syndrome

There are several types of EDS. They can range from mild to life-threatening. Recently, 13 major types of Ehlers-Danlos syndrome have been subtyped. These include:

Classic

Classic-like

Cardiac-valvular

Vascular

Hypermobile

Arthrochalasia

Dermatosparaxis

Kyphoscoliotic

Brittle cornea

Spondylodysplastic

Musculocontractural

Myopathic

Periodontal

Pathophysiology of EDS

Ehlers-Danlos syndrome (EDS) is a heterogeneous group of inherited connective tissue disorders characterized by joint hypermobility, cutaneous fragility, and hyperextensibility.

The collagen defect has been identified in at least six of the many types of Ehlers-Danlos syndrome. The vascular form sometimes referred to as type IV, is characterized by a decreased amount of type III collagen.

It is autosomal dominant and caused by mutations in the COL3A1 gene that result in increased fragility of connective tissue with arterial, intestinal, and uterine ruptures and premature death.

Types V and VI are characterized by deficiencies in hydroxylase and lysyl oxidase, an important posttranslational modifying enzyme in collagen biosynthesis. Type VII has an amino-terminal procollagen peptidase deficiency. Type IX has abnormal copper metabolism. Type X has nonfunctioning plasma fibronectin.

In Ehlers-Danlos syndrome types I and II, the classic variety, identifying the molecular structure in most individuals who are affected is difficult. Causative mutations may involve the COL5A1, COL5A2, and tenascin-X genes and are implied to be in the COL1A2 gene. Nonetheless, in most families with autosomal dominant Ehlers-Danlos syndrome, the disease appears to be linked to loci that contain the COL5A1 or COL5A2 genes. Although half of the mutations that cause Ehlers-Danlos syndrome types I and II are likely to affect the COL5A1 gene, a significant portion of the mutations result in low levels of mRNA from the mutant allele as a consequence of nonsense-mediated mRNA decay.

What causes Ehlers Danlos Syndrome?

Ehlers-Danlos syndromes (EDS) are genetic disorders that can be caused by mutations in several different genes, including COL5A1, COL5A2, COL1A1, COL3A1, TNXB, PLOD1, COL1A2, FKBP14, and ADAMTS2. However, the underlying genetic cause is unknown in some families.

Mutations in these genes usually change the structure, production, and/or processing of collagen, or proteins that interact with collagen. Collagen provides structure and strength to connective tissues throughout the body. A defect in collagen can weaken connective tissues in the skin, bones, blood vessels, and organs, resulting in the signs and symptoms of EDS.

How EDS is inherited?

The two main ways Ehlers-Danlos syndrome is inherited are:

Autosomal dominant inheritance (hypermobile, classical and vascular Ehlers-Danlos syndrome) – the faulty gene that causes Ehlers-Danlos syndrome is passed on by one parent and there’s a 50% risk of each of their children developing the condition

Autosomal recessive inheritance (kyphoscoliotic Ehlers-Danlos syndrome) – the faulty gene is inherited from both parents and there’s a 25% risk of each of their children developing the condition

A person with Ehlers-Danlos syndrome can only pass on the same type of Ehlers-Danlos syndrome to their children. For example, the children of someone with hypermobile Ehlers-Danlos syndrome can’t inherit vascular Ehlers-Danlos syndrome.

Signs, Symptoms, and Complications of EDS

Classical (cEDS)

cEDS (formerly EDSI and EDSII) is associated with the primary problems described above, skin hyperextensibility, joint laxity, and fragile blood vessels. Scars are very thin, discolored, and stretch with time. Such paper-like (papyraceous) scarring occurs especially over prominent bony pressure points such as the knees, elbows, shins and forehead.

Classical-like (clEDS)

clEDS is similar in clinical course to cEDS. Genetic causes of cEDS and clEDS differ.

Cardiac-valvular type (cvEDS)

cvEDS is a rare subtype of EDS wherein patients may have minor signs of EDS with severe defects to their aorta, requiring surgical interventions.

Vascular type (vEDS)

vEDS (formerly EDSIV), can be identified at birth with noticeable clubfoot deformities and dislocation of the hips. In childhood, inguinal hernia (partial slip of intestine beyond the abdominal wall) and pneumothorax (collection of air between the lung and chest wall, impairing proper lung inflation) are commonly experienced and are indicative of this syndrome.

Hypermobility type (hEDS)

hEDS (formerly EDSIII) comes with a defined set of complications to be managed but is generally a less severe form of the syndrome. For example, aortic root dilation is usually minimal and does not significantly increase the risk for dissections. The major complications to patients with hEDS are musculoskeletal in nature. Frequent joint dislocation and degenerative joint disease are common and associated with a baseline chronic pain, which affects both physical and psychological well being.

Arthrochalasia type (aEDS)

aEDS (formerly EDSVII, A and B) is associated with the lifelong risk for the dislocation of multiple major joints concurrently. This condition makes achieving mobility significantly challenging.

Dermatosparaxis type (dEDS)

Patient with dEDS (formerly EDSVIIC) tends to show a set of common body features. These include short stature and finger length, loose skin of the face, with comparatively full eyelids, blue-tinged whites of the eye (sclera), skin folds in the upper eyelids (epicanthal folds), downward slanting outer corners of the eyes (palpebral fissures) and a small jaw (micrognathia). A major complication of dEDS is herniation, the improper displacement of an organ through the tissues holding it in a proper position.

Kyphoscoliotic type (kEDS)

kEDS is accompanied by scleral fragility, increasing the risk for rupture of the white globe of the eye. Microcornea, near-sightedness (myopia), glaucoma and detachment of the nerve-rich membrane in the back of the eye (retina) are common and can result in vision loss. Patients experiencing floaters, flashes or sudden curtains falling across their visual field should seek immediate medical attention.

Brittle cornea syndrome (BCS)

BCS is a variant of EDS that also involves the eyes. People with variant risk ruptures to the cornea following minor injuries with scarring, degeneration of the cornea (keratoconus), and protrusion of the cornea (keratoglobus). Patients may have blue sclera.

Spondylodysplastic type (spEDS)

spEDS, previously spondylocheirodysplastic type, describes an EDS variant with skeletal dysmorphology. It primarily involves the spine and the hands. Clinical presentation can include stunted growth, short stature, protuberant eyes with bluish sclera, wrinkled skin of the palms, atrophy of muscles at the base of the thumb (thenar muscles), and tapering fingers.

Musculocontractural type (mcEDS)

mcEDS is characterized by progressive multisystem complications. This subtype is especially associated with developmental delay and muscular weakness plus hypotonia. Patients often have facial and cranial structural defects, congenital contractures of the fingers, severe kyphoscoliosis, muscular hypotonia, club foot deformity, and ocular problems.

Myopathic type (mEDS)

mEDS is characterized by muscle hypotonia evident at birth with muscles that do not function properly (myopathy). Scoliosis and sensorineural hearing impairment may accompany this condition.

Periodontal type (pEDS)

pEDS type (formerly EDS VII) has findings that include disease of the tissues surrounding and supporting the teeth (periodontal disease), potentially resulting in premature tooth loss.

Diagnosis and Test for Ehlers Danlos Syndrome

A diagnosis of the Ehlers-Danlos syndromes (EDS) is typically based on the presence of characteristic signs and symptoms. Depending on the subtype suspected, some of the following tests may be ordered to support the diagnosis:

Collagen typing, performed on a skin biopsy, may aid in the diagnosis of vascular type, arthrochalasia type, and dermatosparaxis type. People with EDS often have abnormalities of certain types of collagen.

Genetic testing is available for many subtypes of EDS; however, it is not an option for most families with the hypermobility type.

Imaging studies such as CT scan, MRI, ultrasound, and angiography may be useful in identifying certain features of the condition.

Urine tests to detect deficiencies in certain enzymes that are important for collagen formation may be helpful in diagnosing the kyphoscoliosis type.

Is there any treatment for Ehlers Danlos Syndrome?

There is no cure for Ehlers-Danlos syndrome, but treatment can help you manage your symptoms and prevent further complications.

Medications

Your doctor may prescribe drugs to help you control:

Pain. Over-the-counter pain relievers — such as acetaminophen (Tylenol, others) ibuprofen (Advil, Motrin IB, others) and naproxen sodium (Aleve) — are the mainstay of treatment. Stronger medications are only prescribed for acute injuries.

Blood pressure. Because blood vessels are more fragile in some types of Ehlers-Danlos syndrome, your doctor may want to reduce the stress on the vessels by keeping your blood pressure low.

Physical therapy

Joints with weak connective tissue are more likely to dislocate. Exercises to strengthen the muscles and stabilize joints are the primary treatment for Ehlers-Danlos syndrome. Your physical therapist might also recommend specific braces to help prevent joint dislocations.

Surgical and other procedures

Surgery may be recommended to repair joints damaged by repeated dislocations. However, your skin and the connective tissue of the affected joint may not heal properly after the surgery.

Surgery may be necessary to repair ruptured blood vessels or organs in people with Ehlers-Danlos syndrome, vascular type.

Precautions & Prevention of Ehlers-Danlos Syndrome

Genetic counseling can help you understand the inheritance pattern of the type of EDS that affects you and the risks it poses for your children.

Here are a few guidelines you can do to reduce your risk:

Avoid contact sports, weightlifting, and other activities

Use protective gear

Use mild soaps and sunscreen

Use assistive devices to help decrease stress on your joints