No fewer than 18 person reportedly killed in Russia missile strike in Ukraine.


Oleksiy Danilov has praised the Ukrainian military’s efforts in the ongoing war against Russia in Ukraine.

The secretary of the National Security and Defense Council identified as Oleksiy Danilov has praised the Ukrainian military’s efforts in the ongoing war against Russia in Ukraine.

Oleksiy Danilov also commended the weapons, equipment, and logistics supplied to the Ukrainian armed forces by the Western countries for defense and assault.

The Western weapons are already changing the course of the war in Ukraine and they are making an impact as expected, Oleksiy Danilov disclosed.

The Ukrainian military’s artillery weapons are “leveling” the number of Russian troops in Ukraine and we haven’t started yet, the secretary of the National Security and Defense Council, praised Ukrainian artillery accuracy.

The United States and the United Kingdom are the highest suppliers of military logistics, weapons, ammunition, and defense equipment to the Ukrainian armed forces since the Russia-Ukraine war began on February 24th, 2022.


Parasomnias are a group of sleep disorders marked by unusual actions.


Parasomnias are a group of sleep disorders marked by unusual actions, activities, or physiological events that occur during sleep or sleep-wake transitions.

Parasomnias are divided according to the sleep phase in which the symptoms occur, either rapid eye movement (REM) or non-REM (NREM). Symptoms may include simple or complex abnormal movements, such as sleep talking, sleepwalking, sleep terrors, and dream enactment, or emotions, dreams, and autonomic activity. Diagnosis involves a thorough history taking from the individual and anyone who witnesses the movements or behaviors.

In some cases, polysomnography is required; especially if comorbid sleep disorders are suspected. Some conditions resolve over time without any treatment. For those that require intervention, options include lifestyle or risk modifications, different forms of therapy, and medications. Choice of management is affected by individual preferences.


Parasomnias are more often seen in children than in the adult population. In children, the NREM parasomnias are more common than REM parasomnia. Amongst these, the prevalence rate of confusional arousals in children of age group 3 to 13 years is 17.3% and more than 15 years is 6.9%. The prevalence rate of sleepwalking under 12 years of age is 17% and that of sleep terror is 6.5%.

Nightmares are seen in 10% to 50% of the pediatric population. Parasomnias are more frequently seen in children with underlying neurologic and psychiatric issues like epilepsy, attention-deficit hyperactive disorder (ADHD) or developmental issues.

Sleep terrors are much less common in the pediatric population, with estimations as low as 3%. Nightmare disorder affects up to 6% of the population. REM sleep behavior disorder only appears in 1% of the general population, but at least 50% of those with REM sleep disorder suffer from comorbid neurodegenerative disorders, such as Parkinson’s disease, dementia with Lewy bodies, and multiple system atrophy.

Types of Parasomnias

These are the most common types of actions associated with parasomnias:

Non-REM related parasomnias:

Sleepwalking: Once thought to be acting out of dreams, sleepwalking actually takes place during deep sleep (not REM sleep, when dreams typically occur). Sleepwalkers often perform routine activities such as dressing and cleaning. Sleepwalking is quite common in children and affects approximately 1 percent of adults. Bouts of sleepwalking can be triggered by stress, anxiety, excessive alcohol, or epilepsy.

Night terrors: These severe attacks cause people, usually children, to appear to wake up and scream in fear or panic. However, these individuals are typically inconsolable, and tend to have no recollection of the event the following day.

Sleep talking: Sleep talking is when the sleeper mutters isolated speech or full conversations but has no memory of doing so. It occurs in all sleep states and can be associated with other disorders that cause a person to awaken slightly, REM-related parasomnias and sleep apnea.

Sleep-eating disorders: These episodes, like sleepwalking, occur during partial awakenings from deep sleep and cause individuals to eat without any knowledge of what they are doing, or any memory of what they have done.

REM-related parasomnias

Sleep paralysis: People with sleep paralysis are unable to move or open their eyes and this happens just as they are falling asleep or waking up. Things that might increase your risk for REM-related parasomnias include not getting enough sleep, irregular sleep-wake schedules, and stress.

Nightmare disorder: This describes recurrent, intense nightmares accompanied by emotions such as anxiety, anger, or disgust. These nightmares are different from sleep terrors because the patient is able to recall their dreams with great detail and has difficulty going back to sleep. Nightmares are common with acute stress disorder and post-traumatic stress disorder (PTSD), and occur in both non-REM and REM sleep. The nightmares may persist even after other PTSD symptoms have been resolved. This disorder may be treated either with therapy, medication or a combination of both, depending on the severity of the disorder.

Parasomnias risk factors

There are a number of different risk factors that might contribute to parasomnia development. Some of the most common are the following:

Age: Age would certainly be an important factor since parasomnias like bedwetting and sleepwalking often occur during childhood.

Stress: Stress is also a heavy contributor to different types of parasomnias like sleepwalking, night terrors, sleep-related eating disorders, sleep paralysis and more.

Genetics: Additionally, genetics can play a huge role. If your parents struggle with any types of parasomnia, you are most likely to have them as well.

Drug and alcohol abuse: It will not only ruin your liver but also cause a whole bouquet of new parasomnias and definitely worsen any existing ones.

Medicine: No matter how effective your medications are, some of them may have various types of parasomnias as a side effect.

PTSD: Post-traumatic stress disorder (PTSD) is frequently associated with a type of parasomnias like nightmare disorder. Almost ⅘ of PTSD patients would experience nightmare disorder during at least 2-3 months after the trauma.

Causes of Parasomnias

There are many possible causes of parasomnia. The disorder might be associated with multiple triggers, including:





Substance use

Certain medications

Irregular sleep schedules, like shift work

Other sleep disorders, like insomnia

Sleep deprivation

Neurological conditions, like Parkinson’s disease

Symptoms of Parasomnias

Symptoms of parasomnias vary depending on which form you have. However, some common symptoms most parasomnias share include:

Difficulty sleeping

Fatigue during the day

Waking up disoriented

Exhibiting unexplainable behaviours in your sleep

Frequent nightmares

Having bruises and injuries, you can’t explain

Parasomnias complications

Significant complications from Parasomnia (e.g. injury)

Suspected specific conditions

Nocturnal Seizures

REM Sleep Behavior Disorder

Obstructive Sleep Apnea in Children

Periodic Limb Movement Disorder

Diagnosis and test

Your sleep medicine specialist will ask you and your sleep partner about your sleep symptoms. You will also be asked about your medical history, family history, alcohol use and any substance abuse. You’ll be asked about your current medications. You may be asked to keep a sleep diary and your bed partner may be asked to keep track of your sleep events.

Other sleep disorders tests include:

Sleep study (polysomnogram): This is a sleeping laboratory in which you’ll be monitored as you sleep. Your brain waves, heart rate, eye movements and breathing will be recorded as you sleep. Video will record your movements and behavior. While some sleep studies can be done at home, an in-lab study will be recommended if there’s concern for parasomnia.

Video electroencephogram (EEG) or sleep EEG: These tests help your healthcare provider see and record your brain activity during a brain event.

Neurologic exam, CT or MRI scan to detect degeneration of the brain or other possible neurologic causes of your symptoms.

Treatment and medications

Parasomnia treatment depends on the type and severity of the parasomnia. In the cases of frequent or recurring parasomnia, medication can help manage it. Some medications prescribed to help treat parasomnia include:






However, if your parasomnia is caused by medication, your doctor may recommend a different medication or dose.

People with parasomnia may also benefit from cognitive behavioral therapy. This type of therapy often helps with mental health concerns, like stress and anxiety. Other methods that can be used alongside cognitive behavioral therapy include psychotherapy, relaxation therapy and hypnosis.

There are also some treatment options people can try at home, such as:

Scheduling awakenings, where a parent wakes a child about 15 to 30 minutes before they spontaneously wake up. This can help minimize behaviors that follow a certain pattern, such as sleepwalking and night terrors

Creating a safer sleep environment, such as sleeping alone, removing dangerous items from the home, locking doors and windows, placing the mattress on the floor and sleeping with extra padding

Parasomnia episodes can vary from person to person, which is why it’s important to talk to your doctor to receive an individualized treatment plan. Our sleep medicine experts are here to help you get a safe and comfortable night’s sleep.

Prevention of Parasomnias

Although some causes of parasomnias are less likely to be prevented, such as those due to neurological diseases, mental health issues or heredity, others may be prevented by following some of the same management approaches discussed in this article.

These include getting seven to nine hours of sleep a night sticking with consistent bedtime and wakeup times, and limiting alcohol and recreational drug use. Also, ask your healthcare provider to review your current medications. Many can disrupt sleep. If this is the case, perhaps different drugs can be prescribed.


Cyclosporiasis is a parasitic infection in the intestines that can cause symptoms such as watery diarrhea and stomach pain.

What is Cyclosporiasis?

Cyclosporiasis is a parasitic infection in the intestines that can cause symptoms such as watery diarrhea and stomach pain. Cyclosporiasis is most common in tropical or subtropical areas and regions of poor sanitation.

The most commonly involved areas for United States citizens are Mexico, Peru, and Guatemala. Most cases of this form of food poisoning develop in people who have traveled to these areas or eaten produce imported from these countries.

What causes Cyclosporiasis?

Cyclosporiasis is an illness of the intestines caused by a parasite called Cyclospora.

How is Cyclosporiasis spread?

People become infected with Cyclospora by consuming food or water contaminated with feces that contains the parasite. Cyclospora needs to be in the environment for about 1 to 2 weeks after being passed in stool to become infectious for another person. Because of this, it is not spread directly from one person to another.

Cyclospora is found in many parts of the world but is most common in tropical and subtropical regions. Foodborne outbreaks have been linked to various types of fresh produce, including raspberries, basil, cilantro, snow peas, and mesclun and romaine lettuce. No commercially frozen or canned produce has been connected to Cyclospora infections.


Cyclospora species are variably acid-fast, round-to-ovoid organisms that measure 8-10 µm in diameter. Cyclospora species exogenously sporulate and have 2 sporocysts per oocyst. Transmission follows ingestion of oocysts in fecally contaminated water or produce. Direct person-to-person transmission is considered unlikely because the oocysts are not infectious when excreted; the oocysts undergo sporulation outside the human host before becoming infective. The median incubation period is 1 week, during which time the organism invades enterocytes of the small intestine.

Cyclospora species are characterized by an anterior polar complex that allows penetration into host cells, but the life cycle of the parasite and the mechanisms by which it interacts with human host target cells to cause disease are poorly understood.

What are the risks of getting Cyclosporiasis?

Your risk may increase if you eat contaminated fresh foods imported from tropical and subtropical areas where the Cyclospora parasite is common.

Imported foods that have been linked to outbreaks of cyclosporiasis include:





Mesclun lettuce

Snow and snap peas

Pre-packaged salad mix


Some people infected with the microscopic parasite that causes cyclospora infection to develop no signs or symptoms. For others, signs and symptoms — which usually begin within two to 11 days of eating contaminated food or drinking contaminated water — may include:

Frequent, watery diarrhea

Bouts of diarrhea alternating with bouts of constipation

Loss of appetite and weight loss

Bloating, flatulence, and burping

Stomach cramps

Nausea and vomiting

Muscle aches


Fatigue — this symptom may last long after the active infection has gotten better

A general feeling of unwellness (malaise)

Diarrhea may end by itself within a few days, or it may last for weeks. If you have HIV or another condition that compromises your immune system, the infection can last for months if not treated.


The prolonged diarrhea of untreated cyclospora infection can cause dehydration. If you’re an otherwise healthy adult, you can treat dehydration by drinking more fluids. Some people may need to be hospitalized to receive intravenous fluids because they’re at higher risk of severe dehydration. Examples include:

People with other serious illnesses

Infants and young children

Older adults

How Do Health Care Professionals Diagnose Cyclospora Infections?

Cyclospora infections are not common in the U.S., so individuals who have been in endemic areas or who have other health concerns (for example, long-term diarrhea, eating imported foods without careful washing) should inform their medical caregivers if they suspect a Cyclospora infection. In turn, medical caregivers need to alert laboratories they suspect such infections so that the stool samples will be tested specifically for Cyclospora parasites. In addition, it is likely other tests will be done to determine if other pathogens or similar parasites like Microsporidia or coccidian Isospora parasites are causing the symptoms.

Labs may need to examine and concentrate samples of stool specimens because only a low number of oocysts are shed into the feces. In addition, special stains (note that acid-fast staining of the parasite is variable), fluorescence microscopy, or PCR tests are used to find and identify the parasites.

What is the treatment for Cyclospora infections?

Cyclospora infection often goes away by itself, and mild or asymptomatic cases require no treatment. For those who require treatment, the best option is oral trimethoprim-sulfamethoxazole (TMP-SMX, also called co-trimoxazole) (Bactrim, Septra) twice daily for seven to 10 days.

For those who continue to have symptoms or have persistent oocysts on stool examination, another seven-day course is usually effective. Sometimes anti-diarrheal medication is recommended, but only under the doctor’s approval. For people with a sulfa allergy, there are few good options.

There are reported cases where nitazoxanide (Alinia) twice daily was successful as an alternate therapy. One small study suggested that ciprofloxacin (Cipro, Cipro XR, ProQuin XR) twice daily for seven days is an option in adults.

However, it has a higher failure rate compared to TMP-SMX. These medications are not approved for routine use in pregnancy. Pregnant women should check with their obstetrician (ob-gyn doctor) before taking any new prescription.

Lifestyle and home remedies

To prevent or treat mild to moderate fluid loss from severe diarrhea associated with cyclospora infection, it’s generally adequate for healthy adults to drink water.

For children and infants, you may want to use an oral rehydration solution, such as Pedialyte. Sports drinks and carbonated beverages don’t offer the right balance of nutrients that children need.

How can Cyclosporiasis be prevented?

When traveling to a country where Cyclospora is found, you can reduce your risk by:

Avoiding food that has been washed in local drinking water

Drinking water from a safe source


Cooked food

Fruit that you can peel yourself

It can be hard to prevent cyclosporiasis. This is because washing produce does not always get rid of the Cyclospora parasite that causes the illness. You can reduce your risk by:

Following safe food handling practices for production on a daily basis

Cooking produce imported from countries where Cyclospora is found

Consuming fresh produce grown in Canada, especially during the spring and summer months when an increase in non-travel related cases of Cyclospora are reported in Canada.

Lipodystrophy syndromes are rare disorders marked by a lack of body fat.

What is Lipodystrophy?

Lipodystrophy syndromes are rare disorders marked by a lack of body fat just beneath the skin’s surface.

Which type a person has is determined by the patterns of fat loss on the body and whether the disease is acquired or genetic. Due to the body’s inability to process fat, lipodystrophy leads to severe metabolic, hormonal, cardiovascular and fat storage disorders.

Because the disease affects an individual’s outward appearance, it can also have deep psychological effects that may need to be addressed by a mental health care professional.

People who have lipodystrophy can end up with extra fat deposits in their legs, on the face, the back of the neck, or abdomen, and within the liver (called non-alcoholic fatty liver disease). This happens because the body is unable to maintain a layer of fat beneath the skin’s surface, so the fat he or she consumes concentrates elsewhere in the body.

Pathophysiology of Lipodystrophy

The metabolic alterations seen in patients with lipodystrophies result from an overall decrease in adipose tissue burden. Adipose is a metabolically active tissue that has many physiologic functions. Adipose tissue not only provides insulation but also serves to mediate inflammation and secrete many hormones involved in endocrine regulation. In patients with lipodystrophies, a reduction in adipose leads to a deficiency in certain hormones.

In particular, a decrease in leptin has been described in many lipodystrophy patients. Leptin is a proinflammatory adipokine secreted by adipose tissue. It is often referred to as the “satiety hormone” because it regulates mediators of appetite and energy expenditure. A decrease in leptin, whether in the setting of intrinsic leptin deficiency or lipodystrophy, leads to the downstream complications of hyperinsulinemia, insulin resistance with possible progression to diabetes, hypertriglyceridemia, and hepatic steatosis.


The prevalence in adults varies from 2-60% but for UK adults a recent paper quotes a prevalence of 17%. With increased awareness there are now fewer new cases of LDHIV. A prevalence of 33% has been quoted for highly active antiretroviral therapy (HAART) treated HIV children.

Types of Lipodystrophy

The first type of lipodystrophies includes those that are present at birth:

Congenital generalized lipodystrophy (CGL) is one of the most well-studied lipodystrophy syndromes with over 300 reported cases in the literature. Affected subjects have extreme paucity of adipose tissue from birth, leading to a muscular appearance with prominent veins and an acromegaloid appearance, along with severe acanthosis nigricans, hepatosplenomegaly, and early onset of diabetes, hypertriglyceridemia and steatohepatitis. Marked hypoleptinemia is a universal feature of this disease.

Patients with familial partial lipodystrophy (FPL) have normal fat distribution at birth but usually develop fat loss from the extremities after puberty. The well-studied type of FPL is the Dunnigan variety (FPLD) in which fat loss from the limbs and often the trunk, especially anteriorly and over the breasts, is usually accompanied by excess fat deposition over the face and neck. Metabolic abnormalities are similar to those seen in patients with CGL but may not be as severe in some patients. Since the extent of fat loss is not uniform, circulating leptin levels may also vary from low to normal in these patients.

The second type of this disease includes patients who acquire it during their lifetime:

Acquired lipodystrophies may occur in association with other autoimmune disorders or panniculitis, or may be idiopathic. Patients with acquired generalized lipodystrophy (AGL) may show severe metabolic complications similar to patients with CGL, while patients with acquired partial lipodystrophy (APL), also known as Barraquer-Simons syndrome, who invariably have well-preserved lower body fat, have lesser incidences of metabolic complications compared with other lipodystrophy syndromes. Rare forms of acquired partial lipodystrophy associated with autoinflammatory syndromes may however be associated with significant metabolic abnormalities.


Inherited lipodystrophy is a genetic disorder, meaning one or both parents typically pass the abnormal gene to their child. The type of lipodystrophy your child has depends on which genes she or he inherited.

Acquired lipodystrophy is not passed down from parent to child, but develops at some point in childhood, adolescence or adulthood. It can be caused by:


An infection such as measles or pneumonia

An autoimmune disease such as rheumatoid arthritis

The cause of an acquired lipodystrophy can also remain unknown.

Risk factors

Experts do not know the exact cause of many cases of acquired lipodystrophy. Certain medications and autoimmune conditions may increase a person’s risk.

Related autoimmune conditions, beyond those listed just above, and may include:

Autoimmune thyroiditis

Autoimmune hepatitis

Autoimmune hemolytic anemia

Rheumatoid arthritis

Sjogren’s syndrome

Sicca syndrome

The following infections may also increase the risk of developing acquired lipodystrophy:



Infectious mononucleosis

Diphther osteomyelitis



Lipodystrophy symptoms

Lipodystrophy can affect all or part of the body. Signs and symptoms can include areas of the body that appear thin while other areas may appear to be enlarged. Children with the genetic form of lipodystrophy may have symptoms and signs including

Prominent musculature

Protruding belly

Voracious appetite

Early puberty

Cognitive deficiencies

Growth faster than their peers

Increased bone age


Metabolic complications in lipodystrophy can be severe and life-threatening. They are more common in those with a greater extent of fat loss, especially the generalised lipodystrophies. CGL and AGL feature reduced levels of leptin, causing over-feeding which results in the abnormal deposition of fat in other organs with associated metabolic complications, such as:

High insulin levels resulting in insulin-resistant diabetes

Hypertriglyceridemia causing recurrent attacks of pancreatitis

Polycystic ovarian syndrome, infertility, and masculinisation in females

Fatty liver disease, which can progress to liver cirrhosis and eventual liver failure

Enlargement of the liver and spleen

Hypertension and cardiovascular disease.

Other complications include:

Kidney disease

Peripheral T-cell cutaneous lymphoma, occuring in 7% of AGL

Significant psychological distress resulting from the appearance of fat loss

Physical discomfort arising from the loss of fat pads from the buttocks and feet.

Lipodystrophy diagnosis

Doctors typically diagnose lipodystrophy by performing a physical exam and asking about symptoms. They may try to eliminate the possibility of other conditions, such as Cushing’s syndrome or anorexia.

The following diagnostic tests can help support the diagnosis and rule out other conditions:

Genetic tests

Tests to check blood chemical profiles

Whole-body MRI to check for patterns of fat loss characteristic of acquired lipodystrophy

Renal biopsy to check for kidney involvement in cases of suspected APL


Currently, there is no specific treatment that will permanently replace adipose tissue.


The reduced amount of adipose tissue and its impact on appearance is distressing to some people. In certain cases, cosmetic interventions and surgery have been used. The best approach to cosmetic surgery in lipodystrophy has not been formally studied.


The main treatment focus in lipodystrophy is on a low-fat or low energy diet. The aim of dietary therapy is to reduce the amount of ectopic fat in organs which contributes to insulin resistance and the development of metabolic complications of lipodystrophy. Dietary intervention can produce major improvements in many of the established complications that are associated with ectopic fat such as insulin resistance, high blood fat and liver inflammation.


In certain types of lipodystrophy Leptin replacement therapy may be prescribed. Leptin is a hormone which is made by adipocytes (fat cells). Leptin regulates appetite. In patients affected by lipodystrophy leptin levels can be low due to the reduced number of adipocytes and when this happens it may increase appetite. Increased appetite and excess food intake will have a negative effect on metabolism. In such cases Leptin therapy can reduce appetite and improve some of the metabolic complications of the condition. It is most often used in cases of generalised lipodystrophy where there is a near absence of fat.

Psychological support

As a chronic condition and one that is appearance-altering, lipodystrophy can be associated with psychological distress. Some patients may benefit from accessing a variety of psychological support services.


Where specific complications arise such as diabetes and high triglycerides in the blood, medical therapy specific to those conditions may be recommended. It is not uncommon for lipodystrophy patients with diabetes to require high doses of insulin, in that instance concentrated forms of insulin therapy or sometimes insulin pump therapy may be needed. All of these medical interventions are prescribed in conjunction with a low energy/low-fat diet.

Metabolic Surgery

Among patients with partial lipodystrophy and metabolic complications that are not adequately controlled using conventional therapy, metabolic surgery, specifically has been used effectively. Roux-en-Y gastric bypass reduces the size of the upper stomach and thus supports weight loss. In addition, it improves general metabolic problems like reduction of high blood sugar levels and amelioration of insulin sensitivity in muscles, liver and fat tissues.

Prevention of Lipodystrophy

It is important, having identified aetiological factors, to try to prevent LDHIV. Efforts in this direction are being targeted on:

Increasing awareness amongst doctors and patients, together with regular assessment, may help early identification. Early identification may be assisted by techniques such as MRI.

Choice of antiretroviral regimen, avoiding combinations of PIs and certain NRTIs.

Early treatment and interventions for metabolic changes (as these may promote the LDHIV).

General advice on diet and exercise. This may include the use of supplements, high fiber, and omega 3, etc.

Earlier treatment of the HIV infection may help prevent LDHIV (before AIDS develops and before the CD4 count falls below 200/mm3).

Ex-militant leader and head of operations of Darlon Oil and Gas assassinated in Bayelsa state.

Indukapo Ogede, ex-militant leader and head of operations of Darlon Oil and Gas Servicing Limited, has been assassinated in Yenagoa, the Bayelsa State capital.

Ogede was said to have been killed in an execution manner Sunday night at a hotel off Tamic road in Okutukutu, where he usually stays when he is in Yenagoa.

His assailants, armed and wearing military uniforms, were said to have driven into the hotel Sunday afternoon and paid for three rooms ostensibly as a decoy of their real motive.

A source at the hotel said they later went to the beer to drink while waiting for their target to return from an assignment he went to town to execute.

Some close to the deceased at the hotel suspected something was amiss with the behaviour of the suspected assassins and decided to alert him on phone but he did not pick his call as he was driving to the hotel.

Eyewitness accounts indicate that on sighting his car entering the premises, they ran towards him and shot him twice.

The gunshot caused pandemonium in the hotel and the area as the people scampered for safety while the gunmen made their way to escape.

Ogede was rushed to the Glory Land hospital, Opolo where he was confirmed dead and his body moved to the Federal Medical Centre, Yenagoa.

A visit to the scene on Monday indicated that Police detectives have been to the hotel to take questions from the management and those who witnessed the shooting.

The Chairman, Darlon Oil and Gas Limited, Chief Levi Wilson, confirmed the assassination of Ogede in a statement and appealed to the various law enforcement agencies to investigate the killing and bring the perpetrators of the dastardly act to book in a bid to ensure that such incident do not repeat itself in the nearest future.

Wilson described the death as one too many regretting that the deceased left behind his parents, wife and children.

The Bayelsa State Police Command through its spokesman, Asinim Butswat has however appealed for calm stating that the Police are already investigating.


Russia unleashed seven missiles on Odesa Oblast, Ukraine.

According to report, Russian Military has unleashed seven missiles on Odesa Oblast, Ukraine destroying various assets in Ukraine.

Recall that Vladimir Putin announced that the war in Ukraine has not yet started and that Russia will fight till the last Ukrainian following the interference from Western countries especially the United States and the United Kingdom.

The recent seven missile strikes launched by the Russian military reportedly destroyed a house, port infrastructure, and some fields, but there were no casualties, according to Ukraine’s Southern Operational Command.

Russia is known to use deadly weapons, especially missile systems in its war with Ukraine.

One of the deadly weapons used by the Russian military in Ukraine is the Kh-22 missiles, The TOS-1A, a Soviet-era Multiple Launch Rocket System (MLRS) system capable of firing thermobaric warheads, 9K720 Iskander, 3M-54 Kalibr, BM-21 Grad, BM-27 Uragan, BM-30 Smerch amongst others according to Wikipedia.