Merrick Garland US Attorney general said on Thursday he personally approved the dramatic raid on Donald Trump’s Florida home.

US Attorney General Merrick Garland said on Thursday (Aug 11) he had personally approved” the dramatic raid on Donald Trump’s Florida home and, in a highly unusual move, was requesting the warrant justifying the search be made public.

The country’s top prosecutor did not reveal the reason for the unprecedented search of the residence of a former American president, and condemned unfounded attacks on the FBI and the Justice Department that followed it.

I personally approved the decision to seek a search warrant, Garland told reporters. The department does not take such a decision lightly.

The search warrant was authorised by a federal court upon the required finding of probable cause, he said.

While noting that ethical obligations prevented him detailing the basis of the raid, Garland said he had asked a Florida judge to unseal the warrant because Trump had publicly confirmed the search and because of the “substantial public interest in this matter.

Trump has until Friday to contest the request.

The Justice Department motion to unseal the warrant noted – and did not dispute – statements by Trump’s representatives that the FBI was seeking presidential records and potential classified material.

According to US media, the search related to potential mishandling of classified documents taken to Mar-a-Lago after Trump left the White House in January 2021.

The FBI raid on Trump’s palatial Mar-a-Lago residence has sparked a political firestorm in an already bitterly divided country and comes as he is weighing another White House run in 2024.

Trump has condemned Monday’s raid as politically motivated and a “weaponisation of the Justice Department.

Nothing like this has ever happened to a President of the United States before, he said.

In a statement on his Truth Social platform on Thursday, Trump said his attorneys had been cooperating fully and the government could have had whatever they wanted, if we had it.

And then, out of nowhere and with no warning, Mar-a-Lago was raided at 6.30 in the morning, he said, adding that agents even went through the First Lady’s closets and rummaged through her clothing and personal items.

Garland criticised unfounded attacks on the professionalism of the FBI and Justice Department agents and prosecutors following the raid.

I will not stand by silently when their integrity is unfairly attacked, he said.

Men and women of the FBI and the Justice Department are dedicated patriotic public servants.

Leading Republicans have rallied around the former president and harshly denounced the Justice Department and FBI.

US police were engaged in a standoff Thursday with an armed man who tried to break into an FBI office in Cincinnati, Ohio, although there was no immediate indication the event was linked to the raid on Trump’s home.

The Justice Department typically does not confirm or deny whether it is investigating someone.

Garland, a former prosecutor and judge who has a reputation as a stickler for protocol, took pains to emphasize the law was being applied fairly.

Faithful adherence to the rule of law is the bedrock principle of the Justice Department and of our democracy, he said. Holding the rule of law means applying the law evenly without fear or favour.

Since leaving office, Trump has remained the country’s most divisive figure and a force in the Republican party, continuing to sow falsehoods that he actually won the 2020 vote.

On Wednesday, the 76-year-old Trump was questioned for four hours by Letitia James, the New York state attorney general who is investigating the business practices of the Trump Organization.

Trump is also facing legal scrutiny for his efforts to overturn the results of the 2020 election and over the January 6 attack on the US Capitol by his supporters.

Trump was impeached for a historic second time by the House after the Capitol riot he was charged with inciting an insurrection but was acquitted by the Senate.

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Tetralogy of Fallot is a combination of four heart defects that can result in a baby turning blue or cyanotic.


Tetralogy of Fallot is a combination of four heart defects that can result in a baby turning blue or cyanotic because of a lack of oxygen in the blood. It usually is diagnosed in infancy.

The heart consists of four chambers: the two upper chambers, called atria, where blood enters the heart; and the two lower chambers, called ventricles, where blood is pumped out of the heart. Valves that act as one-way doors control the flow between the chambers and between the arteries. The heart also has been pictured as two side-by-side pumps with one side pumping blood into the lungs and the other side pumping blood from the lungs back to the body.

Blood is pumped from the right side of the heart up through the pulmonary valve and the pulmonary artery to the lungs, where the blood is filled with oxygen. From the lungs, the blood travels back down to the left atrium and left ventricle and is then pumped through another big blood vessel called the aorta to the rest of the body.

Tetralogy of Fallot is a combination of four different heart problems that include:

Pulmonary stenosis: A condition where the pulmonary artery (that carries blue blood from the right ventricle to the lungs to be oxygenated) is blocked. Either the muscle below or above the valve, or the valve itself, may be too narrow or underdeveloped, impeding blood flow.

Ventricular septal defect (VSD): A hole between the ventricles, the heart’s two lower pumping chambers.

Overriding aorta: The aorta (the largest blood vessel in the heart, which carries pink blood to the body) is not positioned correctly and straddles both the right and left ventricles just above the VSD.

Right ventricular (RV) hypertrophy: The right ventricle is the lower heart chamber of the heart that pumps blue blood to the pulmonary arteries. Hypertrophy is when the chamber’s muscle wall becomes thickened, where it is normally thin. This occurs in TOF because of the extra effort the muscle exert with TOF anatomy.


TOF is the most common form of cyanotic congenital heart disease. It comprises approximately 3.5% of infants born with congenital heart disease and 8-10% of cyanotic cases. Worldwide reported prevalence of TOF is 0.28 per 1,000 live births. There is no gender predilection.

Pathophysiology of Tetralogy of Fallot

The development of the human heart starts around the 20th day of gestation, with the fusion of the outer endocardial tubes into a single tubular structure, the cardiac tube. Subsequently, the cardiac tube folds and loops, with the development of an atrium that is cranial and dorsal, and a primitive ventricle is moving downward, ventrally, and to the right.

The right ventricle is the dominant chamber in the embryo and fetus, receiving 65% of the venous return, and is the main contributor to the lower part of the body, the placenta, and the lungs. The right ventricle can be described by three components: the inlet, which consists of the tricuspid valve chordae tendineae and papillary muscles; the trabeculated apical myocardium; and the infundibulum or conus.

The exact embryologic process that contributes to the development of tetralogy of Fallot still is unknown, but an association that had been observed is an anterior and cephalad deviation of the infundibular septum that results in a misaligned ventricular septal defect, with an overriding aortic root causing a subsequent right ventricular outflow obstruction.

The ventricular septal defects seen in patients with tetralogy of Fallot are usually perimembranous that can extend into the muscular septum. Different factors can contribute to the right ventricular outflow obstruction, including the pulmonary valve that is usually bicuspid and stenotic, the hypoplastic pulmonary valve annulus, the deviation of the infundibular septum that causes a subvalvular obstruction, and the hypertrophy of the muscular bands in this region.

The degree of the overriding aorta usually varies and receives blood flow from both ventricles. The physiological process surrounding the hypercyanotic episodes or “Tet spells” in tetralogy of Fallot consist of either a decrease in systemic vascular resistance or an increase in pulmonary resistance contributing to a right-to-left shunt across the ventricular septal defect, causing marked desaturation

Tetralogy of Fallot Risk factors

While the exact cause of tetralogy of Fallot is unknown, some things might increase the risk of a baby being born with this condition. Risk factors for tetralogy of Fallot include:

A viral illness during pregnancy, such as rubella (German measles)

Drinking alcohol during pregnancy

Poor nutrition during pregnancy

A mother older than age 40

A parent who has tetralogy of Fallot

The presence of Down syndrome or DiGeorge syndrome in the baby

Causes of Tetralogy of Fallot

Doctors don’t know what causes most cases of tetralogy of Fallot and other congenital heart defects.

Certain conditions or factors that occur during pregnancy may raise your risk for having a child with tetralogy of Fallot. These conditions and factors include:

German measles (rubella) and some other viral illnesses

Poor nutrition

Overuse of alcohol

Age (being older than 40)


Heredity may play a role in causing tetralogy of Fallot. An adult who has tetralogy of Fallot may have an increased chance of having a baby with the condition.

Children who have certain genetic disorders, such as Down syndrome and DiGeorge syndrome, often have congenital heart defects, including tetralogy of Fallot.

Scientists continue to search for the causes of tetralogy of Fallot and other congenital heart defects.

Tetralogy of Fallot Symptoms

Cyanosis is a very common sign. Healthy babies can sometimes also have bluish skin around the mouth or eyes from prominent veins under the skin, but their lips and tongue look pink. Babies who have low oxygen levels in the blood usually have blue lips and tongues in addition to bluish skin.

A child with TOF might have sudden episodes of deep cyanosis, called “hypercyanotic spells” or “Tet spells,” during crying or feeding. Older children who have Tet spells will often instinctively squat down, which helps to stop the spell.

Other signs include:

Heart murmur


Tiring easily

Difficulty breathing

Rapid heartbeat (palpitations)


Clubbing, where the skin or bones around the tips of fingers are widened or rounded


If left untreated, it can cause these problems:

Blood clots (which may be in the brain causing stroke)

Infection in the lining of the heart and heart valves (bacterial endocarditis)

Abnormal heart rhythms (arrhythmias)

Heart failure


Diagnosis and test

Your child’s doctor may have heard a heart murmur during a physical examination and referred your child to a pediatric cardiologist for a diagnosis. In this case, the heart murmur is caused by the turbulence of blood flowing through the obstruction from the right ventricle to the pulmonary artery. Symptoms your child exhibits will also help with the diagnosis.

A pediatric cardiologist specializes in the diagnosis and medical management of congenital heart defects, as well as heart problems that may develop later in childhood.

The cardiologist will perform a physical examination, listening to the heart and lungs and make other observations that help in the diagnosis. The location within the chest that the murmur is heard best, as well as the loudness and quality of the murmur (such as harsh or blowing) will give the cardiologist an initial idea of which heart problem your child may have. Diagnostic testing for congenital heart disease varies by the child’s age, clinical condition and institutional preferences. Some tests that may be recommended include the following:

Chest X-ray: A diagnostic test that uses X-ray beams to produce images of internal tissues, bones and organs onto film.

Electrocardiogram (ECG or EKG): A test that records the electrical activity of the heart, shows abnormal rhythms (arrhythmias or dysrhythmias), and detects heart muscle stress.

Echocardiogram (echo): A procedure that evaluates the structure and function of the heart by using sound waves recorded on an electronic sensor that produce a moving picture of the heart and heart valves.

Cardiac catheterization: A cardiac catheterization is an invasive procedure that gives very detailed information about the structures inside the heart. Under sedation, a small, thin, flexible tube (catheter) is inserted into a blood vessel in the groin and guided to the inside of the heart. Blood pressure and oxygen measurements are taken in the four chambers of the heart, as well as the pulmonary artery and aorta. Contrast dye is also injected to more clearly visualize the structures inside the heart.

Tetralogy of Fallot treatment and medications

Medical treatment

Squatting: Parents may observe the infants squatting or keeping their knees to their chest – this manoeuvre helps increase venous return, therefore increasing systemic resistance. Parents should be advised to put the child in this position whilst awaiting medical review.

Prostaglandin (PG) infusion: This helps maintain PDA in the more severe-extreme forms of TOF and must be started urgently following delivery to avoid the neonate collapsing. Depending on local practice, either PGE1 (alprostadil) or PGE2 (dinoprostone) may be used. Side effects to note include apnoeas, bradycardia and hypotension.

Beta-blockers: propranolol is commonly used in both “tet” spells and prophylaxis in moderate-severe disease. It works by reducing the heart rate thus venous return.

Morphine: Reduces respiratory drive therefore also reduces hyperpnoea

Saline 0.9% bolus can be used in “tet” spells as a volume expander to increase pulmonary blood flow through the RVOTO.

Surgical treatment


Transcatheter RVOT stent insertion: this is sometimes done in the neonatal period for infants with severe-extreme TOF to relieve RVOTO. This is done to buy time until the child is bigger whilst providing a patent passage for pulmonary blood flow.

Modified Blalock-Taussig (BT) shunt: This procedure aims to mimic a PDA and increase pulmonary blood flow before definitive repair. It can be done either by anastomosis of the subclavian artery to the pulmonary artery or by creating an artificial shunt using synthetic material (usually GoreTex). The latter is called a modified BT shunt.

Other alternatives include insertion of an RV to PA conduit (to bypass the RVOTO) and PA banding (to reduce pulmonary overloading, especially in pink TOF with mild PS).

Definitive repair:

Performed under cardiopulmonary bypass via median sternotomy, this involves RVOT stenosis resection, RVOT/pulmonary artery augmentation and VSD patch closure. Depending on the degree of PS, PV is either repaired or spared.

Timing depends on severity of symptoms but usually not performed younger than 3 months old or older than 4 years.

Post-operative care: immediately following surgery, the child will require stabilisation in Cardiac Intensive Care Unit (CICU). Once they are well enough they will be stepped down to the cardiology ward prior to discharge.


No known prevention exists for Tetralogy of Fallot.

Ataxia telangiectasia (AT) is a complex genetic neurodegenerative disorder that may become apparent during infancy or early childhood.

What is ataxia-telangiectasia?

The disorder is characterized by progressively impaired coordination of voluntary movements (ataxia), the development of reddish lesions of the skin and mucous membranes due to permanent widening of groups of blood vessels (telangiectasia), and impaired functioning of the immune system (i.e., cellular and humoral immunodeficiency), resulting in increased susceptibility to upper and lower respiratory infections (sinopulmonary infections).

Individuals with AT also have an increased risk of developing certain malignancies, particularly of the lymphatic system (lymphomas), the blood-forming organs (e.g., leukemia), and the brain.

In those with AT, progressive ataxia typically develops during infancy and may initially be characterized by abnormal swaying of the head and trunk. As the disease progresses, the condition leads to an inability to walk (ambulation) by late childhood or adolescence.


The ATM gene encodes the protein kinase ATM, which is the key regulator of cellular response to double-strand breaks (DSB) in DNA. Therefore, ataxia-telangiectasia symptoms include all the possible consequences of the perturbations in DNA damage response (DDR).

One basic defect associated with the malady is the abnormal sensitivity of ataxia-telangiectasia cells to x-rays and certain radiomimetic chemicals but not to ultraviolet irradiation, which leads to chromosome and chromatid breaks.

Breakpoints are randomly distributed, but nonrandom chromosome rearrangements selectively affect chromosomes 7 and 14 at sites that are concerned with T-cell receptors and heavy-chain immunoglobulin coding and with the development of hematologic malignancies. Such disturbances could account for the frequency of infections and neoplasias.

As has been shown by Guerra-Maranhao et al, ataxia-telangiectasia patients are at high risk of having impaired responses to infection with pneumococci, which may be one of the causes of recurrent sinopulmonary infections in these patients.

The authors analyzed the production of antibodies to polysaccharide antigens in patients with ataxia-telangiectasia and found that the levels of immunoglobulin G (IgG) antibodies to serotypes 1, 3, 5, 6B, 9V, and 14 of Streptococcus pneumoniae before and after immunization with 23-valent polysaccharide vaccine were significantly lower than in a healthy population.

Causes of ataxia telangiectasia

AT is caused by alterations (mutations) at specific areas within an individual’s genetic information. Each of us has a large amount of genetic information that is organized into smaller segments known as genes. Genes provide the necessary instructions that our cells need to perform different functions within our bodies.
AT develops when there are alterations in a specific gene known as ATM, located on chromosome 11 at position q22.3. The ATM gene has the ability to produce an enzyme called a “serine/threonine kinase” that has several important functions:

It acts as a tumor suppressor the enzyme keeps cells from growing and dividing too quickly and it promotes cell death.

It interacts with other proteins when DNA damage occurs following exposure to ionizing radiation.

The ATM enzyme coordinates DNA repair by activating other proteins that are essential for the repair process to occur.

It is believed that through a combination of these mechanisms, the ATM enzyme aids in the prevention of specific types of cancer, such as leukemia and lymphoma.

Patients with AT carry an alteration in both copies of the ATM gene in all the cells of their body. When both copies of the ATM gene are altered within the cells of an individual with AT, the altered gene copies will make less of the ATM protein or an ATM protein that does not function properly.

As a result, cells are hypersensitive to radiation and instead of repairing damaged DNA, the defective ATM proteins allow for alterations to accumulate in other genes because effective DNA repair is unable to occur. When this happens, individuals with AT are at an increased risk to develop leukemia and lymphoma. Additionally, altered ATM genes may allow cells to die inappropriately, particularly in the cerebellum. This can result in the neurological symptoms that occur in AT.

Risk Factors

Since A-T is an inherited genetic disease and symptoms often start in early childhood, lifestyle risk factors, or things you can change to lower your risk of developing a particular disease, aren’t applicable.

But having A-T raises the risk of developing cancer. Reducing lifestyle risk factors for cancer is even more important for people with the condition and their family members.

People with A-T have about a 40% risk of developing cancer, most likely leukemia or lymphoma. Those two cancers account for 85% of all cancers in people with A-T.1 People with A-T also have an increased risk of developing breast cancer, ovarian cancer, stomach cancer, skin cancer, and bone and soft tissue cancer.

The American Cancer Society says being a carrier of the ATM gene (inheriting one abnormal copy of the gene) has been linked to a high rate of breast cancer in some families. A 2021 review study identified a specific mutation or ATM variant associated with increased breast cancer risk called the V2424G mutation.

General risk factors for cancer include:

Older age

A personal or family history of cancer

Using tobacco



Some types of viral infections, such as human papillomavirus (HPV)

Specific chemicals

Exposure to radiation, including ultraviolet radiation (UV) from the sun

Symptoms of ataxia telangiectasia

Symptoms include:

Decreased coordination of movements (ataxia) in late childhood that can include ataxic gait (cerebellar ataxia), jerky gait, unsteadiness

Decreasing mental development, slows or stops after age 10 to 12

Delayed walking

Discoloration of skin areas exposed to sunlight

Discoloration of skin (coffee-with-milk-colored spots)

Enlarged blood vessels in skin of nose, ears, and inside of the elbow and knee

Enlarged blood vessels in the whites of the eyes

Jerky or abnormal eye movements (nystagmus) late in the disease

Premature graying of the hair


Sensitivity to radiation, including x-rays

Severe respiratory infections that keep coming back (recurring)

What are possible complications of ataxia telangiectasia?

Other problems that may occur with A-T include:

Weakened immune system

Increased risk for leukemia

Increased risk for lymphoma

Extreme sensitivity to radiation such as from X-rays

Pulmonary disease, growth hormone deficiency, and diabetes

Higher risk of leukemia and lymphoma

How is ataxia telangiectasia diagnosed?

A-T is suspected whenever a child develops signs of ataxia, meaning unsteady walking. Testing of the ATM gene is available, and genetic mutations can be identified in, about 90% of people with A-T. The following tests may be more helpful in determining if someone has A-T or another type of ataxia:

Immunoblotting (ATM protein testing). This is the best test to diagnose A-T. Nearly all individuals with A-T will have very low or no amounts of the protein made by the ATM gene.

Radiosensitivity assay. Since people with A-T have an increased sensitivity to radiation, removing some cells and treating the sample with radiation therapy can help make the diagnosis. It can take up to 3 months to get a result from this test.

ATM kinase activity. This test looks at the activity level of the protein made by the ATM gene. Little to no activity means that there is likely a mutation in the ATM gene.

How ataxia telangiectasia is treated?

Treatment for ataxia-telangiectasia (A-T) is geared toward preventing and managing symptoms. Common treatments include fighting respiratory infections and controlling the spread of and severity of dilated blood vessels.

Home Remedies and Lifestyle

A person with A-T should stay out of the sun, as direct sunlight causes worsening of the widened and dilated blood vessels in exposed areas (telangiectasia).

Nutritional therapies are being looked at for their potential in reducing symptoms associated with A-T including nutritional deficiencies and immune system dysfunction.

A particular nutritional supplement, myo-inositol (also called inositol), has been looked at with respect to its impact on blood sugar management and insulin sensitivity. Inositol is currently used in treating metabolic disorders like diabetes.

Diabetes is a common co-occurring disorder in people with A-T. Inositol may be useful for reestablishing balance in people with A-T who have type 2 diabetes or insulin-resistant diabetes.

Postural Drainage

Respiratory infections can make it even more challenging to clear the airways. Postural drainage is an at-home remedy that can help.

Postural drainage is when you put your head at a lower level than the rest of our body so your bronchial tubes and lungs can drain. You can do this by lying on your back, bending your knees, placing a pillow under your knees, and resting there until you feel relief.

Vitamin E Therapy

Vitamin E is an antioxidant that’s said to protect cells against free radical damage caused by things like environmental toxins, smoke, and pollution. In A-T, a person’s cells are under increased oxidative stress. For people with A-T, vitamin E may be used to provide temporary relief of symptoms.


Prescriptions for A-T vary widely depending on the symptoms being treated. Prescriptions for A-T are used off-label, meaning there is no approved medication for A-T treatment.

Off-label prescriptions include certain anti-Parkinson’s and anti-epileptic drugs. Research on their usefulness in treating symptoms is limited due to small sample sizes.

Amantadine sulfate is one example. It may help in the treatment of motor symptoms (movement issues) in A-T. One study on 17 children treated with amantadine sulfate found that over 76% of patients had at least 20% symptom improvement after eight weeks.7

Other examples include:

Baclofen for ataxia-related muscle spasms or cramps

Clonazepam or Neurontin (gabapentin) and Lyrica (pregabalin) for dizziness and vertigo, tremors, and rest tremors

Prozac (fluoxetine) for speech and swallowing, and for uncontrolled laughing and crying


Metformin lowers blood sugar levels (acting as an anti-hyperglycemic agent). It’s recommended as first-line therapy for type 2 diabetes, which can be a complication associated with living with A-T.

Valium: Valium (diazepam) can be used to help symptoms of slurred speech and involuntary muscle contractions.

Antibiotics: Antibiotics can be used to treat the respiratory infections common in persons with A-T.

Immune-Heightening Medications

Researchers are working in clinical trials to investigate how the following immune-heightening medications may help treat immune-related problems associated with A-T. To date, none have been approved in the treatment of A-T or A-T-specific symptoms.

Examples include:

Levamisole: This experimental cancer drug was pulled from the U.S. market in 2000 due to adverse side effects.

Interleukin-2: This drug regulates white blood cell activity involved in immune response.

Intron (interferon): This drug interferes with your immune system environment in such a way that it signals antiviral activity. It is FDA approved for treating certain cancers and hepatitis B.

Chemotherapeutic agents (e.g., cyclophosphamide): These suppress your immune system.

Antilirium (physostigmine): This drug works on the nervous system to alter signals between nerves and muscles.

Surgeries and Specialist-Driven Procedures

A-T can cause difficulties in drinking, chewing, and swallowing, and tremors can make holding utensils and eating particularly challenging. Treatments for such problems should be advised by a speech-language pathologist.

Dietitians can also help with specific changes like increasing calories and discussing the benefits and drawbacks of certain food supplements.

Gastronomy Tube

To prevent nutritional deficiencies and aspiration, a gastronomy tube (G-tube) can be inserted through the abdomen skin and into the stomach. With early placement, nutritional supplementation allows growth, improves stamina, and decreases the risk of lung damage from aspiration.6

G-tubes are recommended in the following situations:

A child cannot eat enough to grow.

A person of any age cannot eat enough to maintain weight.

Aspiration is problematic.

Mealtimes are stressful or too long, interfering with other activities.

Physical and Occupational Therapy

Physical therapy can be used to help with muscle strength and prevent tightening and shortening in the limbs.

Physical therapy, as well as speech therapy and occupational therapy, can be useful in maintaining certain functions, but cannot prevent their eventual progressive decline

Neurological deterioration eventually leads to needing to use a wheelchair and other adaptive devices. Children and teens may need adaptive equipment to attend school.

Genetic Counseling

Genetic counseling has been said to potentially be of benefit to people with A-T and their families. It can provide education for the patient and family members regarding the possibility and potential consequences of genetic testing for A-T relatives, including siblings. Genetic counseling may also be useful for families seeking support in interpreting results.

How can prevent ataxia telangiectasia?

Since ataxia telangiectasia is the result of a genetic mutation, there is no way to prevent it from occurring. You can take steps to reduce your risk of having a child with a genetic condition by avoiding smoking and avoiding exposure to chemicals. If you plan on becoming pregnant, talk with your healthcare provider about genetic testing to understand your risk of having a child with a genetic condition like ataxia-telangiectasia.

Nigeria: No fewer than six persons killed several others injured in herdsmen attack in Benue state.

Armed Fulani herdsmen on Wednesday night killed six persons and left several others with gunshot and machete wounds in Logo Local Government Area (LGA) of Benue State.

The incident occurred at Tse Ngojov and Tse Valem Yaweh of Mbagum community in Tombo council ward of Logo LGA of the state at about 9 pm.

Paul Hembah, Special Adviser to Governor Samuel Ortom on Security who confirmed the incident to Daily Independent on Thursday noted that the Council Chairman, Salome Tor had earlier briefed him on the incident.

He explained that as sad as the incident was, Governor Ortom has directed that the wounded be moved to the Benue State University Teaching Hospital (BSUTH), while arrangements should be made, preparatory to the burials of the dead.

Daily Independent sources in Logo LGA said those that lost their lives to the attack were Ander John, Vihior Mtomga, Morgan Varem, Saaior Uyoosu, Aondoyima Tarbo and Peter Asondo.

He said that those that were injured include Igbaule Valem, Chiater Tarbo and Mrs Terngu Tyavkase Use.

According to him, the injured persons were currently receiving treatment at the Primary Health Care (PHC) facility at Ayilamo in Logo LGA.

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China ramps up military treath to bring Taiwan under it control.

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Taiwan says it has dispatched planes and ships to the area to monitor the situation.

Taiwan has scrambled fighter jets to protect its territory after China deployed more than 30 warplanes to buzz the island.

Dramatic video shared by Chinese state TV shows missiles being launched and jets mid-flight as part of joint military operations in the waters surrounding Taiwan.

For its part, Taiwan’s Ministry of Defence has shared its own footage of military exercises, including the second day of live-fire artillery drills in Pingtung, the southernmost province on the island.

The defence ministry claimed that 10 Chinese warships and 36 military aircraft had been detected around Taiwan on Wednesday.

In response, the ministry said it was monitoring the situation and deploying its own naval vessels, aircraft, and land-based missile systems.

Earlier on Wednesday, sources claimed Chinese naval ships were active off both the east and west coasts of Taiwan, while fighter jets were also flying close to the halfway point of the Taiwanese straight.

Sharing a clip of its fighter jets taking over ahead of journeys over the Taiwan Strait, Taiwan’s defence ministry said: “We are at the ready keeping our country safe. We stand on the frontlines to protect our country from any threat.

We neither seek escalation nor instigate disputes. Maintaining the status quo is our solemn mission, it went on, over footage of military exercises.

Holding the median line, safeguarding our seas and our sovereignty, we have never given in to challenges, and never will, the video concluded.

In response, China has shared its own footage of the Eastern Theatre Command of missile launches and military jet takeoffs and mid-air refuellings, as well as navy ships on so-called drills around Taiwan.

The video shared by state broadcaster CCTV also included a computer-generated animation of Chinese missiles landing in four distinct zones in the waters surrounding the island.

US warships are being deployed to the region, USNI reports, raising fears of a broader international conflict.

We will continue to stand by our allies and partners, US undersecretary of defence for policy Colin Kahl said.

So even as China tries to chip away at the status quo, our policy is to maintain the status quo with a free and open Indo-Pacific which is what I think most of the countries in the region would prefer.

China’s military announced on Wednesday it has completed various tasks around Taiwan, but will continue to conduct regular patrols.

Theatre forces will keep an eye on the changes in the situation in the Taiwan Strait, continue to carry out training and preparation for combat, organise regular combat readiness patrols in the direction of the Taiwan Strait, and resolutely defend national sovereignty and territorial integrity.

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Nigeria: Governor Nasir El-Rufai has write Muhammadu Buhari that terrorists are creating parallel Government in Kaduna state.

According to report, terrorists are consolidating their grip on communities in Kaduna with a parallel government and permanent operational base in the North-western state near Nigeria’s capital Abuja, Governor Nasir El-Rufai has told President Muhammadu Buhari.

Mr El-Rufai warned the president of the terrorists’ consolidation in a late July memo.

The terrorists belonging to Ansaru al-Musulmina fi Bilad al-Sudan, or Ansaru for short, are believed to have moved to Birnin Gwari in Kaduna State in 2012 when they broke away from Boko Haram.

According to intelligence reports and human sources consulted in further reporting for this story, the terrorists that formed Ansaru were responsible for some of the high-profile attacks claimed by Boko Haram before the split.

Such attacks included the UN building bombing of August 2011 and the kidnap of some foreigners. According to the sources, Ansaru pledged allegiance to Al-Qaeda in the Islamic Maghreb, AQIM, in 2020, and is responsible for many of the high-profile abductions as well as armed attacks on the police in Kaduna State.

But researchers and negotiators with an understanding of the working of the terrorists told PREMIUM TIMES that a collaboration of the terror groups, including the original Boko Haram – also called JAS -, ISWAP, Ansaru, and bandits, is evolving.

In 2021, 1,192 people lost their lives in Kaduna State due to banditry, terrorism, communal clashes, violent attacks, and reprisals.

In the first six months of 2022, 645 people lost their lives in such circumstances across the state, the government said.

In Mr El-Rufai’s late July memo, he lay bare how the terrorists have infiltrated and dominated communities and formed “a parallel governing authority, exercising control over social and economic activities and dispensation of justice in the area.

He said the terrorists have advanced in their plans to make Kaduna forest areas their “permanent operational base” for the North-west region, citing a series of intelligence reports.

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Observed movement patterns and intercepted communications of migrating terrorists have shown a clear interest in setting up a base, with the stretches of forest area between Kaduna and Niger states strongly considered,” he wrote.

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Nigeria: INEC deny recognizing Apkabio and Ahmed Lawan as Senatorial candidate.

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Taiwan holds artillery live drill to text combat readiness.

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